Polyneuropathy code according to ICD 10. Treatment of diabetic polyneuropathy

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Diagnostics: yak viyaviti hvorobu

Whether the pathology of the nerve fiber was trivial for an hour, it could be asymptomatic. І for diabetics, і for chemically injected, і navіt for wicked alcohol will take an hour, so that the consistency of fabrics is needed.

Lyudin may periodically experience discomfort when walking, poking around in the area, or some other symptoms that are usually driven by the infection. The patient is taken to an appointment with a neurologist, even if the symptoms are manifested more often and more complexly.

Among the instrumental methods of diagnostics, we are more precise for the differentiation of pathology and electroneuromyography. The equipment for її conduction, please, check the sign. The procedure for the patient is absolutely safe and painless. An alternative method is used only in special neurological settings. Zastosovuyut also ultrasound of the nerve with dodatkovy dynamic tests. In addition, it is necessary to carry out visual inspection, signifying clinical analysis, x-ray.

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Differentiate neuropathy from the onset of illness:

1. Vertebrogenic radiculopathy;
2. gonarthrosis;
3. Raynaud's syndrome;
4. Post-insult failure.

types of signs

Symptoms of neuropathy lie without the median topics of pathology, i.e. In one way, when the robot Nervus femoralis is a one-time operation, the muses will collapse on a frequent basis, so in the club-transverse area, the presence is alternative to innervation. And the axis in the area of ​​the pathology is manifested by yaskravish, paresis of the chotirigol meat. Patients present such skargi:

1. Weakness in the lower picture, the leg is "not to be heard";
2. Difficult when walking, especially if it is necessary to go down or go down to descend;
3. The sensitivity of the curvatures of the stegna is reduced;
4. Vazhko or ill-advised bigati.

A patient with neuropathy is easy to recognize on the street for a change in movement. For people who suffer from mono - or polyneuropathies are characteristic of chovgannia, not oversubstantiation. The stench to go yak bi "slightly rozkidayuchi" legs, namagayuchis їkh do not zginati.

Yak likuyut pathology

Remarkably, the treatment of the stygonal nerve neuropathy is a trivial process and not always successful. It’s obvious, it’s a complex move, like it’s going to be in front of you, the reason for getting sick.

Drug treatment is represented by vitamins, analgesics, which regenerate and use drugs. The regular course of physiotherapy is of great importance. In case of neuropathy, toxic etiology of liquefaction, they are straightened in the first place in the middle of the country for the release of high-pressure injections. It’s like a robot in high-minded minds, and it’s good to remember that a patient will take a trivial hour if it’s a drug, it’s possible, if it’s necessary to replace it with a less toxic analogue.

The mode and the way of life of the patient is of great importance in the treatment of any pathologies, neuropathies, including. Healthy calls - the best prevention. Tse axiom.

ICD-10 was introduced into the practice of protecting health in the entire territory of the Russian Federation in 1999 by the order of the Ministry of Health of Ukraine dated 27.05.97. No. 170

A new look (ICD-11) is planned by WHO in 2017 2018 roci.

Іf changes and additions of the VOOZ rr.

Processing and transfer zmin © mkb-10.com

Diabetic polyneuropathy code according to ICD-10

Polyneuropathy is a complex of ailments, which includes multiple lesions of peripheral nerves. It is most often necessary to move into the chronic stage and by the volodya with the lower path wider, so that the process of collecting the extra fibers and step by step is going to take all the big bugs.

Classification of polyneuropathy according to ICD 10 є officially known, but not for individual specific features, and does not describe the tactics of treatment.

Symptoms and diagnostics

A clever picture in the first place to be grounded on the ruined robots of the support-and-meat apparatus and the heart-vascular system. Patient skarzhitsya on pain in the muscles, their weakness, judgment and the appearance of the building to normal oversaturation (paresis of the lower symptoms). Frequent frequency of the heart rhythm (tachycardia), symptoms of arterial grip, hypothermia and headaches through changes in the tone of the brain and improper blood circulation of the organs of the central nervous system are given to the backward symptomatology.

When the health of the ailing ointment is gone, I will atrophy, the lyudin basically lie, but it is negatively signified on the grating of the soft tissues. In some cases, necrosis develops.

A handful of remedies for goiters to listen to all of the patient's skargi, to take a look around, to reconsider the tendon reflexes and the sensitivity of the pupils for the help of special tools.

Laboratory diagnostics of blood is effective in terms of the presence of specific pathologies and the reasons for the development of the main disease. It is also possible to increase the adjustment of glucose concentration in non-toxic spores, salts of important metals.

І of the modern instrumental methods for the reduction of electrical neuromography and biopsies of the nerve.

lykuvannya

The International Committee has developed a system for the treatment of polyneuropathy. In the first place, there is an infusion of the head causative factor - to become feigned by organisms for the help of antibiotics, to compensate for the impairment of the endocrine system and hormonal therapy, to change the mind of robots, to change the form of being involved in new alcohol intake, to become operative.

In order to unify the development of an accelerated development, a high-calorific value of the day (at a time of day), a complex of vitamins and minerals, which introduce the robot of the immune system and the trophism of a cell, is indicated.

For the knowledge of symptoms, there are no painful drugs, antihypertensive drugs, as well as muscle stimulants.

For an ailment such diabetic polyneuropathy: code for MKH-10, clinical picture and methods of treatment

Polyneuropathy is a complex of ailments, which includes the so-called multiple lesions of peripheral nerves.

The disease is encouraged to move into the so-called chronic form and the long way wider, so that the process of combining the enemy itself with other fibers and, by and large, overflows into more large spikes.

This pathological camp, called diabetic polyneuropathy ICD-10, encrypts and grows in fallowness as a result of walking, overcoming ailments on the onset of the group: ignition and polyneuropathies. So also diabetic polyneuropathy according to ICD?

So tse take?

Polyneuropathy is the so-called acceleration of the diabetic diabetes, the whole essence of the polyagus is in the generalized accelerated nervous system.

Nerve reduction in polyneuropathy

Call yourself out to show yourself through a significant interval of an hour, which has passed at the moment of diagnosis of damage in the robotic endocrine system. More precisely, the disease can appear in twenty five rockets in the form of development of problems with the insulin in people.

Ale, bully vipadki, if a sickle of a bullet has been detected in patients of endocrinologists already through five years since the moment of revealing pathological diseases of the lump. Rizik zvoriti - the same for children with diabetes, as the first type, so and the other.

Causes of the blame

As a rule, in case of a trivial interruption of the disease and the frequent consumption of the tsukru, the breakdown of the metabolic processes in all organs and systems of the body is diagnosed.

Moreover, the first suffering is the nervous system itself. As a rule, nerve fibers to liven up blood-bearing judges.

We are trivial in pouring in carbohydrates because it is called the destruction of nerves. As a result of which stench falls into the camp of hypoxia, as a legacy, the primary symptoms of illness appear.

With a slight interruption and frequent decompensation, it is significant to accelerate the apparent problems with the nervous system, which step by step inflate a nonvolatile chronic character.

Diabetic polyneuropathy of lower symptoms according to ICD-10

The same diagnosis most often smells the children who are suffering from diabetes.

Denmark of ailment is hostile to the organism of todi, if the peripheral system and fibers are destroyed. There can be provocations by the very different factors.

As a rule, people of the middle age were strong before, in the first place. Most of the time, some people get sick more often. It is also obvious that polyneuropathy is not common in children of preschool and adulthood.

Diabetic polyneuropathy, code for MKH-10, which is E10-E14, invoke enemies of the very top and bottom of people. As a result, responsiveness, praiseworthiness decreases, the quality becomes asymmetric, and blood circulation also decreases. It seems to me that the head particularity of the whole ailment is that those who are guilty are widening in every way, hostile to the very same nerve fibers. It is not marvelous for those who are calling, for those who suffer from their feet.

signs

Sickness, manifested overwhelmingly on the lower symptoms, volodya great number of symptoms:

• feeling a strong oniminnya in the legs;
• swelling of the feet and nig;
• unbearable pain and prickly vision;
• weakness in meat;
• increase or decrease in sensitivity of pictures.

The cutaneous form of neuropathy leads to the following symptoms:

1. diabetic at the first stages. Vaughn is characterized by some of the lower cues, a lot of pokryuvannya and strong writing in them. The presence of ice is smudged in the feet, gomilkovy soblobnyh slopes, as well as in litkovye ointments. As a rule, at night the symptoms become more severe and swelling;
2. diabetic at lower stages. With the appearance of such troubling symptoms: unbearable beating in the lower symptoms, as it can also appear in the country of calm, weakness, atrophy of the muscles and the symptoms of the pigmentation of the school. We will proceed with the development of the sickness, the stink will burn, as a result of which the stench will become more delicate, or will atrophy. Also, in a sick person, a diabetic foot is called so called: it means to grow in size, there is flatness, ankle deformity and neuropathic bulges develop;
3. diabetic encephalopolyneuropathy. For some of the following signs are characteristic: how not to go through severe headaches, instantaneous and increased stomachache;
4. toxic and alcoholic. Їy characteristic symptoms such as severe swelling of the symptoms: sudomy, oniminnya nig, not impaired sensitivity of the feet, weakening of the tendon and reflexes of the muscles, change to the bluishness of the scalp, I will curl on the bluishness of the hair of the hair, change As a heritage, the trophies take off and the swelling of the nig.

diagnostics

Oskilka one type of dosage cannot show again the picture, then diagnostics of diabetic polyneuropathy is carried out using the ICD-10 code for additional decals of popular methods:

As a rule, the first method to follow-up is to take a detailed look at a number of people: a neurologist, surgeon and endocrinologist.

The first doctor to deal with the development of specific symptoms, such as: blood pressure in the lower symptoms and sensitivity, the presence of all the necessary reflexes, conversion to the increase in the size of the machine.

As far as laboratory dosages are concerned, they include: analysis of blood plasma, glucose concentration in blood plasma, cholesterol, as well as the level of toxic speech, which can be found in the body when it is suspected of having toxic neuropathy.

And the axis is instrumental diagnostics of the presence of diabetic polyneuropathy in the organism of a patient according to ICD-10 May on the basis of MRI, as well as electroneuromyography and biopsies of nerves.

lykuvannya

It is important to memorize, but lіkuvannya is guilty of being complex іmіshanim. Vono, it is not uncommon to include in itself perennial drugs, which are direct on the entire process of development.

Even more importantly, the treatment included the intake of these drugs:

1. vitamini. Smells of guilt come into the body at once with food products. The workers use the transport of impulses along the nerves, and also block the negative effects of glucose on the nerves;
2. alpha-lipoic acid. Won the purchased zukru in the nerve tissue, activating the singing groups of enzymes in the cells and changing the nerve damage;
3. antibiotic preparations;
4. іngіbіtory aldose reductase. The stench to stand at the plant is one of the number of transformations of zukru in the blood, which change by itself and infuse on the nerves of the end;
5. actovegin. Winning glucose vikorizannya, will dye microcirculation of blood in arteries, veins and capillaries, which nudge nerves, as well as transferring nerve cells;
6. Calcium and Calcium. At the same time, the power of change of judgment and oniminnya in the pictures of the people may cause the power of change;
7. antibiotics. You can only get it right, if there is a risk of gangrene development.

In addition, as the very form of diabetic polyneuropathy ICD-10 is revealed, the eligible doctor recognizes a professional treatment, as I increase the symptoms of the disease. At the same time, it is possible to help on the outside of the vil_kuvannya.

Even more importantly, it is important to reduce the amount of blood in the blood, and only then to start testing for diabetic polyneuropathy according to the ICD. If you do not kill, then all efforts will be absolutely ineffective.

It is even more important in the case of toxic forms of increasing alcoholic drinks and after being able to take advantage of the suvorian children. A liquid drug is obviously guilty of special medications that cause microcirculation of blood and overload blood clots. Even more importantly, get out of the mess.

Video by topic

Candidate of Medical Sciences about polyneuropathies in diabetics:

You can be intelligent with the help of information, you can see it in the statistic, diabetic neuropathy can be done well. Naygolovnishe - not starting the process. The ailment can be very swirling of symptoms, as it is important not to mention it, so with a reasonable approach to it, you can end up quickly getting confused. For the first symptoms of anxiety, it is important to go through medical procedures, to confirm the transfer of diagnosis. As soon as it goes, you can go to the treatment of ailment.

• Stabilizu rivn tsukru nadovgo
• Introduce the revolving to the insulin in the slurry plant

Treatment of diabetic polyneuropathy

Succinic diabetes є we will broaden illnesses in the whole world. Most often, it is most common to develop chronic diseases. One of the accelerated disease is diabetic polyneuropathy.

Chronic diabetic (sensorimotor) polyneuropathy - the form of neuropathy is broadened, as it is super-susceptible to sensitive, vegetative and ruffled disorders.

E 10.42 diabetic polyneuropathy in type 1 diabetes,

E11.42 diabetic polyneuropathy in DM 2,

G 63.2 Diabetic distal polyneuropathy.

Diabetic polyneuropathy is associated with pain syndrome and often decreases the level of life of patients.

The development of a sickness can lead to more serious acceleration. Such, yak: ataxia, Charcot's angle, diabetic foot syndrome, diabetic osteoarthropathy.

Diabetic polyneuropathy of children can be brought to gangrene and other amputations.

To that, it is important to remember the development, and it is more effective to pick it up even with the first signs of a child with a baby with diabetes.

cause development

The main ethiological factors that provoke the development of diabetic polyneuropathy are taken into account:

1. Courinnya and alcohol;
2. Failure to control blood glucose;
3. vik;
4. Arterial vise;
5. Destruction of spіvvіdnoshennya lіpіdіv (fatty words) blood;
6. Decreases against insulin in blood;
7. Trivial overbig of a diabetic.

The number of up-to-date ones should be taken into account for those who constantly control the level of glucose and the arterial grip, but rather reduce the development of pathology. And the time spent on insulin therapy will reduce the risk of development by a factor of two.

symptoms

Symptoms of diabetic polyneuropathy appear as more symptoms in the lower symptoms. Pekucha, stupid or sverblyachі bіl, rіdsche gostry, prickly і pierced. The most frequent winners are in the foot and to feast on the evening hour. Nadal, more can be manifested in the lower third of the woman's hands.

Patients skarzhitsya on the part of the m'yaziv, pain in the slopes, the ruined walk. Tse is tied with a rosette in the nervous system. Temperamental sensitivity is involved, trophism may appear.

Hvory vidchuv discomfort comes from doty ogyagu. Pain syndrome in such vipadas is of a permanent character and sutta pogirshuє in the back of the self-esteem of the sick person.

Yak viyaviti and clarify the diagnosis?

Diagnosis of polyneuropathy is to be repaired from a doctor's account, which can help you collect anamnesis and a sign of the need for a diagnosis.

Electroneuromyography appears in the capacity of the main preliminaries. In addition, there can be stasosovuvati doslіdzhennya VKSP (vegetative shkіrnyh sympathetic potential).

lіkuvannya pathology

To set the diagnosis for diabetic polyneuropathy, use ethiotropic therapy. It is important to normalize glucose instead of blood glucose. For continuous control, in 70% of cases, more responses change. In some cases, insulinotherapy is indicated.

In the scheme of treatment for oxidative stress, for renewal of edema, it is necessary to use medicinal preparations with an antioxidant effect. By taking the drug, take courses to finish a trivial hour. At tsei period ide control and caution for the patient.

For pain relief, analgesics and protivopalny drugs are used. Ale, as I mean fakhivtsi, I will increase the stink of the odd odor from most of the seen, that trivially, you can get the correct robot into the slurry.

For symptoms of chronic neuropathic pain, anesthetics, antidepressants and antiepileptic drugs are prescribed. In addition to preparations, it is recommended to use vikoristovuvati plasters with lidocaine, gels, ointments and cream.

As a consolidation of a complex treatment of diabetic polyneuropathy, in the presence of a patient, I mean:

• fizio likuvannya,
• magneto and phototherapy,
• electropharesis i strumi,
• electrostimulation of myaziv,
• golkovkolyuvannya,
• hyperbaric oxygenation,
• monochromatic information optimization.

Likuvannya by folk zasoba is allowed to be deprived of a likuyuchiy lykar for the season. As an addition to the traditional methods of treatment, you can use phytherapy and treatment of ointments.

Effective treatment of diabetic polyneuropathy takes into account the individual's condition to the skin patient with a complex of conservative methods of treatment.

What is the value of the treatment of polyneuropathy according to ICD-10?

The polyneuropathy code according to ICD-10 will be different for fallowness, depending on the type of disease. Given the uncomfortable mind of a pathological camp, with any hostilities, a nerve in the body of a man. It also manifests itself in the form of parallels, paresis, problems with trophic tissue and vegetative type growths.

Axonal polyneuropathy of the lower symptoms or of the lower parts of the body develops when the communication processes are broken. For example, an affliction can be manifested when mish, mercury, lead and other words get into the body. In addition, the alcoholic form can be included until the end. For the interruption of polyneuropathy, there is a hostility, pidgostroy, chronic, recurrent.

See the onset of the axonal polyneuropathy:

1. 1. Gostra form. To develop with a stretch of decilkoh dib. The nerve cut is tied with a strong intoxicating organism through the infusion of methyl alcohol, mish'yak, mercury, lead, foul gas and other spoluk. This form of pathology can be no more than 10 dB. Therapy is carried out under the control of a doctor.
2. 2. Subacute. Vaughn grows out with a stretch of decіlkokh tizhnіv. It is typical for toxic and metabolic species. Oduzhati viyde only through the clump of miscellaneous.
3. 3. Chronic. It grows in a stretch of a trivial advance for an hour, for more than 6 months. Such a type of pathology is progressing, as there is a lack of vitamin B12 or B1 in the body, and also lymphoid, cancer, pukhlina, and diabetic diabetes develop.
4. 4. Recurrent. There can be a lot of sick people more than once, and it can be manifested by a stretch of rockets, albeit periodically, and not permanently. It is often possible to achieve this in case of alcoholic forms of polyneuropathy. Qia ailment vvazhaє to be even more insecure. Growing up, only when Lyudin lived on a lot of alcoholic beverages. At the same time, it is not only the amount of alcohol that plays a great role, but also its quality. Tse is disgustingly pouring into the zalny camp of the healthy people. During the period of therapy, it is categorically to abstain from living alcohol. Due to the alcoholic content, it may be necessary to use it as a matter of course.

The demiєlіnіzuyucha form is characteristic of the Bari-Gin syndrome. Tse pathology of the ignition type. Її provoke ailments, infections. With a lot of people, there is an operative type of pain in the legs and weakness in the muscles. The price of the characteristic features of the ailment. After the weak health, symptoms of the sensory form of the ailment appear in an hour. The development of the whole ailment can be carried out in months.

If the patient has diphtheria-type polyneuropathy, then in a couple of days there will be enemies of the cranial nerves. Through the tse of awesome mova, people are important to grow, kovtati їzhu. The integrity of the phrenic nerve will also deteriorate, so it is important for people to dichati. Parallich of kintsivok insisted only through the month, and the whole hour the sensitiveness of the hands would be ruined step by step.

Also, there is a classification of polyneuropathy for provocative officials:

1. 1. Toxic. The qia form is manifested through the detachment of the organism by the chimneys of the chimneys. It may not be just a mish'yak, mercury, lead, ale and a pobutova chemistry. In addition, the toxic form manifests itself with trivial alcoholic deposits in chronic forms, as it is badly infused into the camp of the nervous system and cause malnutrition in the robotic organs of children. Another type of toxic polyneuropathy is diphtheria. Vaughn manifest itself as an accelerated pislya diphtheria. Zazvychay to grow up to do the trick at the grown-ups. The pathology is characterized by small changes, which are linked to the functions of the nervous system. For example, it’s easy to lose weight with the cleanliness of fabrics, and it’s awesome for rukhov’s function. Lіkuvati similar polyneuropathy is guilty of only lіkar.
2. 2. Ignite. Tsey razvidovy zvoryuvannya develop only for the development of ignition processes in the nervous system. At the same time, there is an inappropriate vision, it is in the legs and hands. Maybe the building is ruined and talked about. If any of these symptoms appear, it is necessary to immediately return to the hospital.
3. 3. Allergic. The qi form is developed on aphids of toxicity with methyl alcohol, mish'yak, foul gas and phosphorus-organic fluids. A great role is played by the chronic form of intoxication with the help of others. Unfriendly є prognosis for diabetics, diphtheria and avitaminoses. To achieve the often allergic form of getting sick grows through the trivial implantation of any medication.
4. 4. Traumatic. Tsey rizvid z'yavlyatsya through the removal of serious injuries. Symptoms will only be shown by the onset of the onset of deciduous movements. Name the head symptom є deterioration of the rukhovaya function. Even more important before the hour of exercise є exercise and exercise therapy.

Vidіlyayut and inshi, less widening of the form of polyneuropathy.

Neuropathy nig: diabetic, alcoholic, peripheral, sensory, toxic

Whether it’s a neurological illness, it’s like a neuropathy of the lower symptoms, connected with nerves, so that it’s not possible to get rid of the nerves and sensory nerves. This can lead to a weakening or a loss of sensitivity, and also to the loss of the health of the muscular fibers, it starts to tighten up, so that the locomotion of the musculoskeletal apparatus is improved.

Code for MKH-10

Epidemiology

In patients with diabetes, the frequency of neuropathy of the lower symptoms is 60%. І CDC statistics indicate the presence of peripheral neuropathy in 41.5 million Americans, which is higher in 14% of the US population. Such numbers may seem unrealistic, but the National Institute of Diabetes doesn’t mean that almost half of the patients now don’t know if they have a pathology, which is why the development of ailments is at an early stage of discomfort. lykarya.

According to the assessments of experts, peripheral neuropathy appears in 20-50% of HIV-infected patients and in 30% of oncological patients in chemotherapy.

Spadkov neuropathy Sharko-Mari-Tuta won 2.8 million people in the whole world, and the frequency of Guin-Barré syndrome is 40 times lower, as many malomas are diagnosed.

The incidence of alcoholic neuropathies (sensory and motor) varies from 10% to 50% alcoholics. Ale, if the electrodiagnostic methods are victorious, then neurological problems with the legs can be found in 90% of patients with trivial alcoholic deposits.

Causes of neuropathy of lower symptoms

In modern neurology, there are some of the most common causes of neuropathy of lower symptoms, such as:

• injuries, in case of any malice of the hand, or of the malleable fixation (with splints, tires), they can fix the vice without the need for the hand on the nerves;
• stenosis (zvuzhennya) of the spinal canal, in which there is a stovbur of the spinal nerve, as well as the compression of the ventral nerves, or the heating of the nerve roots;
• craniocerebral trauma, stroke, swelling of the brain (in the first place in the zones of the extrapiracytic system, the cornsus and the pedicle nuclei);
• іnfektsії, vklyuchayuchi viklikaєtsya vіrusom Varicella zoster gerpetichny mієlіt, Gієna-Barre syndrome (rozvivaєtsya at urazhennі vіrusom herpes IV type pіdrodini Gammaherpesvirinae), difterіyu, hepatitis C, hvoroba Lyme (klіschovy borelіoz) SNІD, leprosy (sprichinyuєtsya bakterієyu Mycobacterium leprae), menіngoentsefalіt rіznoї ethology;
• metabolic and endocrine discomfort - diabetes of both types, porphyria, amyloidosis, hypothyroidism (lack of hormones in the thyroid gland), acromegaly (excess of hormone growth);
• autoimmune ailments: rheumatoid arthritis, systemic cervonium vovchak, rosy sclerosis (with ruptured myelin nerves), hospitality roscience encephalomyulitis;
• recessionary illness: neuropathy of Sharko-Mari-Tooth, neurodegenerative ataxia of Friedreich, recessive sphingolipidosis or Fabri ailment; another type of glucogenesis (Pompe's ailment, which is a defect in the lysosomal gene for the enzyme maltasi);
• ailments of motor (rukhovyh) neurons - amyotrophic bichny sclerosis;
• subcortical atherosclerotic encephalopathy with atrophic serpentine speech in the brain (Binswanger's ailment);
• multiple maloma or multiplex plasma cell miloma (in case of malignant degeneration of plasmatic B-lymphocytes);
• Lambert-Iton syndrome (for example, in case of malignant cancer of the leg), neuroblastoma. In some types of neuropathies, it is called Paraneoplastic;
• systemic vasculitis (inflamed blood-bearing vasculature), which can provoke the development of a vascular periarthritis from the deterioration of the innervations of the lower symptoms;
• exchange and chemotherapy;
• toxic infusion of ethyl alcohol, dioxin, trichlorethylene, acrylamide, herbicides and insecticides, mis'yak and mercury, important metals (lead, taliyu and in.);
• In addition, anti-tuberculosis drugs in izonicotinic acid, proto-dominal drugs in the group of hydantoin, fluoroquinolone antibiotics, hypolipinoxydemic statics
• lack of levels in the organisms of cyanocobalamin and folic acid (vitamins B9 and B12), which leads to the development of funicular myelosis.

Riziku factories

Physicians unanimously consider the factors of development of neuropathy of lower symptoms of weakening of immunity to the factors of development of the neuropathy, which infuses on the basis of the organism of bacterial and foreign infections, as well as the decline (the disease).

In addition, the development of adipose diabetes with obesity and metabolic syndrome; rosy sclerosis - diabetes, intestinal problems and pathology of the thyroid gland.

The factors of risk of systemic vasculitis of HIV infection, virus of hepatitis and herpes, sensitization of the organism of the developmental ethology. And the plasma cell mієloma grows easier in the quiet, who will lose their vagu or alcoholic stagnation.

In a large number of cases of blood loss to the brain, necrosis of his tissue, if the myelin sheath of nerve fibers is inadequate, leads to degradation. The first such camp can be spared in patients with cerebrovascular problems.

In principle, all overwhelmed disease can be brought down to factors that cause a neurological breakdown of ruddy functions.

pathogenesis

The pathogenesis of neurological problems with the legs is one of the reasons for this. Physical injuries can be superficially compressed by the compression of nerve fibers, which transforms their health, stretching, through which their integrity is destroyed.

The pathological effect of glucose on the nervous system is not understood, however, with trivial transposition of glucose in the blood, the failure of the conduction of nerve signals along the nerve nerves is an unsettled fact. And with an insulin-dependent diabetes mellitus, it is not just a breakdown in carbohydrate metabolism, but the functional lack of bagatose endocrine valleys, which is poured into the backward exchange of speeches.

Pathophysiological neuropathy in case of Lyme ailments is in two versions: Borrelia bacteria can provoke an immune-mediated attack on the nerve or even without a middle ear of the cell with its toxins.

In the development of amyotrophic lateral sclerosis, the main pathogenetic role of the substitution of dead nerve neurons from the brain structures by the nodes of the glial cells is the main pathogenetic role.

In the pathogenesis of demilinating neuropathies (in the middle of the most common diseases, peroneal amyotrophy of the disease occurs, or Sharko-Mari-Tooth ailment), genetic impairment of the synthesis of Schwann cells of the speech fibers - the sheaths of the nerves in 75% Extend on the nerve along all the way (behind the blame of small, non-localized universities), the myelin sheath of the cell's nerves. Without it - through the degenerative changes of axons - the transmission of nerve signals will break down or be pulled back. In the case of Sharko-Mary-Tooth ailments (due to the lesions of the small nerve, which transmits the impulses to the peroneal muscles of the lower muscles, the spine of the foot), mutations appear on the short arm of chromosome 17 (MFNP222 geni).

Bagatorazova mієloma hostile to the germinal center of the lymph node B-lymphocytes, destroying the proliferation. This is the result of chromosomal translocation between the important lancer genome and immunoglobulin (in 50% of specimens - on chromosome 14, at the q32 locus) and an oncogene (11q13, 4p16.3, 6p21). Mutations lead to dysregulation of the oncogene, and a chubby clone grows and produces abnormal immunoglobulin (paraproteine). And how to vyroblyayutsya with a lot of antibodies to produce up to the development of amyloidosis of peripheral nerves and polyneuropathy in viglyadi paraplegia nig.

Mechanism of detachment with mish'yak, lead, mercury, tricresyl phosphate polyagi in addition to pyrovic acid in the blood, impairing the balance of thiamin (Vitamin B1) and reduced enzyme activity of cholmeninesterase (nerve loss) Toxins provoke a cob drop in the muscle, which triggers an autoimmune reaction, which manifests itself in the swollen muscle fibers and glial cells with further destruction.

In case of alcoholic neuropathy of lower levels of acetaldehyde, there is a decrease in intestinal absorption of vitamin B1 and a fast rate of tiaminpyrophosphate coenzyme, so that the process is reduced to breakdown. So, there is a growing rin of lactic, pyrovine and d-ketoglutaric acids; to go down the glucose uptake and decrease the amount of ATP needed for the training of neurons. In addition, alcoholics have experienced a systemic nervous system at the level of segmental demyelination of axons and loss of muscle at the distal ends of the nerves. They play a significant role in metabolic efficiency of liver failure, associated with alcoholism, spine, lack of lipid acid.

Also alcoholic polyneuropathy

One of the inheritances of chronic alcoholism is polyneuropathy. Dane ailment, according to the International Classification of ailments, code G62.1 and is characterized by impaired functions of the peripheral nervous system. There is an alcoholic polyneuropathy of the lower symptoms at the lower stages of the formation of fallowness due to the implantation of alcoholic beverages and a poor prognosis. The development of an impairment of waste alcohol and other toxins on the structural elements, the storage base of the peripheral nervous system, and the breakdown of the communication processes in them.

Pathological changes occur not only in the cranial and spinal nerves, but also in the brain and spinal cord. It is worth noting that medical statistics show that another skin patient with alcoholism is susceptible to symptoms of polyneuropathy, moreover, women are more likely to get sick, less people.

cause

Alcoholic polyneuropathy develops into a link with a dental infusion of ethyl alcohol.

Regular alcoholic malignancies are known to cause nerve impulses to the point of failure. Surrounded by the price of reliance on exchange processes and outbreaks in the transport of lively conversations. In addition, acetaldehyde, which is ruined and peripheral, and the central nervous system, makes its contribution to the development of ailments.

There is not a lack of vitamins in group B, and the deficiency is established through the deterioration of the process of getting lively words in the small intestine in patients with chronic alcoholism. Chronic alcoholism is susceptible to a disease in the immune system, such as the formation of antibodies, which degrades the nerve tissue. To complement the picture of liver dysfunction symptoms. As a result, the poisonous infusion of alcohol has a number of overwhelmed diseases, which cause the development of alcoholic polyneuropathy.

symptoms

Alcoholic polyneuropathy is diagnosed, as in a sick person's presence such signs of an ailment:

• baking pains in homosexuals and feet;
• myazovi spasms;
• seeing "goose bumps";
• weakness in the lower pictures;
• a decrease in sensitivity in the hands and feet of nig, to accommodate at night;
• "Shlyopati" course;
• blіdі shkіrnі pokrivi;
• ruinous ravnovagi;
• colder gomіlok i feet with normal blood flow;
• empowerment;
• parts of sechovipuskannya;
• one of the hands;
• blue for marmurism of shkiri nig;
• weak tendon reflexes, no more or less visibility;
• parallel or paresis nig;
• change of hairline of homilok;
• trophіchnі virazki;
• hyperpigmentation of shkiri.

diagnostics of illness

The general picture of an affliction, described in ICD 10, does not allow polyneuropathy to be diagnosed at once, some of the symptoms presented may indicate the manifestation of these health problems. With a kind of classy picture, Mayut:

• diabetic polyneuropathy (ICD code 10 - G63.2);
• chronic ignition polyneuropathy (ICD code 10 - G61);
• Hyun-Barre syndrome (code of the International Classification of ailments - G61.0);
• recession of the peripheral nervous system.

When staging a diagnosis, it is necessary to exclude the manifestation of recessive pathologies, inheritance of intoxications (including lykarska), whose symptoms may be similar to the picture. I especially respect the food that comes, not the presence of diabetic or uremic polyneuropathy. For the whole, MRI, nerve biopsy, computer tomography, electro-neuromography and a secondary neurological examination are prescribed.

Diabetic abnormal uremic form can be diagnosed only behind additional biopsies of the nerve. Before the hour of obstezhennya, it is necessary to establish the reasons, which led to the development of the ailment.

Lіkuvannya alcoholic polyneuropathy

When a sign of ailment appears, it is necessary to see it as an alcoholic drink, to see children and to eat a lot of vitamins. At the same time, if the ailment is to continue the disease, then there is no way to find the most effective drugs, not to stop the development of the disease. Therapy includes medication and non-medication treatment.

Medication therapy on the basis of internalization or internal administration of drugs. Preparations include:

• vitamini group B;
• antihypoxanths;
• pentoxifyline, liquids for microcirculation;
• neuromedin for accelerated nerve conduction.

Dodatkovo can be used drugs for zenyatty zapalennya (diclofenac, nimesulid), antidepressants, non-narcotic analgesics, hepatoprotectors. For patients with severe pain syndrome, use carbamazepin or amitriptyline.

In case of non-drug treatment, complex rehabilitation is stagnant, in order to rely on the importance of carrying out physiotherapeutic procedures:

• electrostimulation of the spinal cord;
• magnetotherapy;
• massage;
• electrostimulation of nerve fibers;
• lykuvalnoy physical culture;
• holcoreflexotherapy.

forecast

It is worth noting that medical statistics become ill, there is an uncomfortable prognosis: a greater number of ailments spend on patronage and disability (group II). In the wild, with the speedy beefing up to the factions and the demise of all attributions to the countries, it can be reduced. In medical practice є diseases, if the disability in such ailments was known through the sign of the improvement of health. At the same time, drinking alcoholic polyneuropathy is a process of triviality and vimag of a sickly significant volovych zusil.

prophylaxis

Prevention of alcoholic polyneuropathy of transmission of vicious factors, health impairments of the nervous system. First for everything, the price is put before the implantation of alcoholic beverages. Only if the alcohol is taken into account, there is a 100% guarantee not to fall ill with alcoholic polyneuropathy.

ICD code 10 - diabetic polyneuropathy

Diabetes is not safe due to rapid acceleration, one of which is polyneuropathy. Diabetic polyneuropathy is code according to ICD-10, therefore, it is possible to get sick from marks E10-E14.

chim is not safe

The pathology is characterized by lesions of the group of nerves. Patients with diabetics have polyneuropathy є accelerated when they are interrupted.

Change your mind about the development of polyneuropathy:

• senior clerk;
• bobbing a wag;
• lack of physical activity;
• permanently adjusted glucose concentration in blood.

Neuropathy develops through those that trigger organism, the mechanism of vivennya in carbohydrates, in conjunction with a consistently high concentration of glucose. As a result of the process, structural changes in neurons are observed, and the speed of the impulses is improved.

Diabetic polyneuropathy is classified by ICD-10 yak E10-E14. The whole code is entered into the protocol when the patient gets sick.

symptoms of pathology

Most often diabetic polyneuropathy is hostile to the lower children. Symptoms can be divided into two groups - cob symptoms and signs. It is characteristic of the ear of the ear:

• almost a light pokolyuvannya in pictures;
• onіміння ніг, especially when I go to sleep;
• the loss of sensitivity of expressions.

Often, patients do not pay respect to the symptoms on the cob and go to see if they see more signs:

• post_yni more nig;
• weakening of the muscles of the foot;
• change of tovshchini nigtiv;
• deformation of the foot.

Diabetic polyneuropathy, which is assigned the code E10-E14 according to the ICD, brings a lot of discomfort to the sick and is fraught with serious acceleration. The pain syndrome does not change at night, so the disease is often supervised by sleeplessness and chronic self.

diagnostics

Diagnosis is put on the display of the last picture of the patient's skarg. Necessary additional manpulations:

• perevirka vise;
• reversal of the heart rhythm;
• arterial grip of kintzivok;
• analysis for cholesterol level.

It is also necessary to recalculate the concentration of glucose in the blood, hemoglobin and insulin. For all the analyzes, a patient needs to undergo a complex examination by a neurologist, which is to assess the steps of improving the nerves of a child.

The ICD code E10-E14 in the protocol of the patient's illness means the diagnosis of diabetic polyneuropathy.

lіkuvannya pathology

Lіkuvannya polyneuropathy in a complex approach. For addiction to stagnate:

• drug therapy;
• normalization of glucose concentration in blood;
• progrіvannya nіg;
• physical culture.

The drug therapy is aimed at the improvement of the walls of the nerves, the improvement of the performance and the reduction of the nerve fibers. In case of the establishment of a pathology, it is also necessary to have a miscellaneous therapy, which is aimed at processing the disease and minimizing the risk of introducing infection into the wound.

In the office of exercise therapy, the patients show the right way, as it is necessary to show the day.

An important stage in the treatment of diabetic polyneuropathy is the reduction of glucose concentration in the blood. Gradually adjustments to the tsukru stimulate the rapid development of the improvement of the education, so it is necessary to continue the correction of the sick person.

may riziki

Polyneuropathy (code for MKH-10 - E10-E14) is not without serious acceleration. Deterioration of sensitiveness can lead to the appearance of a great number of trophic virazoks, contaminated with blood. It’s not possible to get sick all the time, you can amputate urazhenoї kіntsіvka.

forecast

With an important mind of a successful result є a quick beast to a licar. Itself on its own the diabet becomes a serious rizik for a sick person's life, for that to listen to the vlasny organ_ism - the most important zavdannya skin patient.

Nowadays it is possible to allow the development of polyneuropathy to the polyneuropathy of children. It is even more important to continuously control the concentration of sugar in the blood to detect a relapse.

Diabetic polyneuropathy of lower symptoms (signs, like likuvati)

The middle of the acceleration of diabetic diabetes is one of the most disliked and very important to bear - diabetic polyneuropathy. Through the struggle of the nerves of the ailments, the blasphemy of the mouths, the feet bother about the jgut, you can see it, the strong sverbinnya, the trivial gostra. Tsі vіdchuttya disgustingly known by antihistamіnnye zasob that simple zealous. As a rule, symptoms will occur at night, ailments can practically be relieved of normal sleep, so depression, panic attacks, mental breakdown can be achieved before polyneuropathy.

On a part of diabetic polyneuropathy, a third of all neuropathies are seized. Immovability of a diagnosis of an accelerated rate of being overwhelmed by diabetes: with a 5-year experience, neuropathy is diagnosed in a skin patient, life with a diabetic is 30 years of age for 90% of children.

Also diabetic polyneuropathy

The destruction of carbohydrate and other types of metabolism in case of sugar diabetes is negatively infused into the entire nervous system, repairing it from the brain and ending with it. The education of the central nervous system was called diabetic encephalopathy, peripheral - diabetic neuropathy.

• sensory - for impaired sensitivity;
• motor - for the worries of nerves, such as service staff;
• I am vegetative, if I get nervous, as a service to the organ of the people.

Sensorimotor neuropathy is the most common type, it is most often to be repaired on the most recent types of the central nervous system and on the links, as a rule, on the lower pictures. This is called distal, from the Latin disto - like. Zvychay snakes are repaired at once on both legs, the stench is also progressed symmetrically. The distal symmetric sensorimotor neuropathy is called "diabetic polyneuropathy", due to the widening of the middle of neuropathies, in the first place, up to 70% of the peripheral nerves are affected.

In such a rank, diabetic polyneuropathy, it is assumed that the damage to the motor fibers of skeletal muscles, mechanoreceptors of the skeletal muscles, tendons, pain receptors, and in the case of diabetic diabetics on the back of the child is taken.

Polyneuropathy is one of the main factors in the development of diabetic foot syndrome, in case of nerve damage, infection is achieved and, as a result, the symptoms appear to be glib, nasty jerks.

Type of diabetic polyneuropathy

See 3 types of diabetic polyneuropathies:

1. Sensory type. Reworking sensitive peripheral nerves, which are nerve fibers of varying diameter, which collect information about our perceptions and transmit them to the brain.
2. Motor type. Stronger ruining of ruffian nerves, which are required for the transmission of information to the messages about the need for speed and relaxation.
3. Zm_shaniy type. In the body, all the nerves work smartly: they are sensitive, they start to feel hot, and they carry the command to pull back their hand, to remove the senses. Rush nerves can be most often complex, for that sensor-motor polyneuropathy - nayposhirenish.

cause sickness

The development of polyneuropathy is without a mediocrity in the form of a sick person's blood glucose for diabetics. It has been clinically reported that more often in the blood of a diabetics a high tsukor is spared, and that more and more people will progress all the acceleration, including polyneuropathy. Even though blood glucose is stably in the norm, after 15 years of illness, signs of polyneuropathy reestablish only in 15% of patients, and all of them are in a mild form.

The reasons for the reduction of nerve cells in the minds of hyperglycemia:

1. Disruption of metabolism.

• Chronic hyperglycemia of the organism of vikoristovuvati is the best way to utilize glucose, when accumulated sorbitol and fructose are accumulated, including in the nerve cells and around them. From the end of the guard, the sheath of the nerve, yak takes care of the fate without the middle in the transmission of impulses;
• glycation of nerve cells;
• ruinuvannya їх shells by violent radicals;
• shortage of muscle in the nerve through blocking the transport of myoinositol.

1. Poshkojennya sudin. Through the diabetic microangiopathies, the judges are guarded to live the peripheral nerves.
2. Slump. The instability to diabetic polyneuropathy is revealed. There is a tribute, that some people have the nerve to go through a few rockets for diagnostics of diabetes, and without such acceleration to live ten years, unimpressed on a high tsukor.
3. Destruction in immunity is the very low-latency reason. In the version, polyneuropathy can be provoked by antibodies to factors in the growth of nerves, such as the organism of the sick person itself.

Symptoms and signs

In case of polyneuropathy, perish, bend over to the sensitive fibers, then to fix the fight of the ruffs. Most of the first symptoms occur on the feet, and then step by step widen on all the lower symptoms, the hands are thirsty in front of them, and in important cases, they live and the breasts.

Strengthening sensitiveness, inability to see from ancient dotypes or odyagu. Goosebumps, onіміння, surface bіl in the camp calm without reason. It is uncharacteristic for the reaction of an organism to a mock, for example, itching when touching.

Weakened sensitivity. Ailment with diabetic polyneuropathy ceases to perceive earlier speech: shortness of the surface when walking barefoot, more when standing on other objects. The water temperature will collapse because the water temperature is too hot, the ice is hot to build up.

Diabetic distal polyneuropathy

Find nerves fibers in the body of people and rots in the legs. Shkokojennya їх on whether dіlyantsі means loss of functions of a nerve, that polyneuropathy is most often є distal, so it is localized in lower images. The most sensible changes are promoted in the so-called "zone of shkarpets" - on the feet and hands. In the first place, tactile, temperamental, more painful sensitivity will break down here.

It is necessary to fix the changes in the sounds, as a result of which the call of the foot changes - to go out and know one on one toe, to restrain the stars. Shkira, relieved by her sensitivity, is becoming a vidminous task for young children, as through the other problems the food and products in the metabolism gradually cease to be hungry; Permanently locally fired ruinu cistkovy fabrics. As a result, the distal polyneuropathy can turn into gangrene and osteomyelitis with the second state of self-sufficiency.

Dіabetichna polіneyropatіya nizhnіh kіntsіvok on pochatkovіy stadії Got takі symptoms yak onіmіnnya, pokolyuvannya, tyazhkіst the feet Vnochi, nemozhlivіst vіdchuti easy dotik, postіyne vіdchuttya cold in his fingers nіg, znizhennya potovidіlennya the feet abo, navpaki, postіyno vologa shkіra, shelled i pochervonіnnya in mіstsyah rubbing.

Yak likuvati polyneuropathy in diabetics

Treatment of diabetic polyneuropathy of lower symptoms at the first stage є reaching a permanently normal blood flow. It has been brought that good control over glucose is brought to regression of the recently diagnosed neuropathy and the generalized mind of the effective treatment of important forms of discomfort.

For the normalization of glucose level in the blood, it is necessary to consult a competent endocrinologist, who can identify a new therapy regimen, in order to obtain more effective symptoms. Because of the sick person at the whole stage, it is necessary to follow the recommendations of the fakhivts, which were formed independently from the licars' preparations, to include the occupation of physical culture and the environment in the harvesting - you will see the result

Likuvannya without drugs

Polyshiti blood circulation, and hence, і harchuvannya tissue in the feet, it is possible with the help of simple non-drug methods. A few times a day it is necessary to work out a light self-massage of the feet. Yakshcho shkira is overdried, with a massage, it is viscous to vikoristovuvati a liquid cream. Grylki and hot baths are fenced off through security, as a diabetic with polyneuropathy may not be aware of, so as a receptor on the surface of the screen.

In any case, it is impossible to interconnect the activity. Obov'yazkovo pidlyagaє walks and skinny day, even with a lot of stitching, but the legs are not overstretched.

For the improvement of blood circulation, the brown awkward complex will be right:

1. Sit down on the glass.
2. Bend - bend your fingers.
3. Viconize the feet of the circles of the hands in the small sides.
4. Pull the socks towards yourself - up to yourself.
5. Roll your feet on round objects - bags, pipes, rocking chair.

In physiotherapy rooms, for pain relief, there are signs of electrophoresis, paraffin therapy, ultratonotherapy, radon and water baths.

RCHRH (Republican Center for Development of Health Protection of the Ministry of Health of the Republic of Kazakhstan)
Version: Clinical protocols of the Ministry of Health of the Republic of Kazakhstan - 2016

Ignition polyneuropathy, unspecified (G61.9), other igneous polyneuropathy (G61.8)

Neurology

General information

Short description

caught
Unified committee for the quality of medical services
Ministry of Health Protection and Social Development of the Republic of Kazakhstan
from "29" falling leaves 2016 to rock
Protocol No. 16

For chronic heat dementia polyneuropathy (CIDP) In the Danish hour, all the drops of firing demilinizing polyneuropathies, which may be larger than the ear and chronically (after 2 months), are extended, which are characterized in some cases by flaring and warning signs.

Specialized codes ICD-10 and ICD-9

Classification

Classification
The clinical classification of the CVDP (Working group for the CVDP EFNS / EPNS 2010) with the views of the typical and atypical options is presented below.

klinichna classification CIDP

Diagnostics (outpatient)

DIAGNOSTICS ON THE AMBULATOR RIVNI

diagnostic criteria
skargi:
· On the weakness in the distal or proximal views nig, inodi asymmetric;
· Oniminnya and paresthesia in the hands and feet;
· On instability when walking;
The first to be inviting to urazhayutsya lower prints - they couldn’t walk, walk along the gatherings, walk from the crystal, win the fall; when the top ideas are obtained, it is difficult to get difficult when the kitchen stuffings are corroded, laces are tied up, and objects are buried.

anamnesis: The neurological symptomatology of the build-up is more often, often with a prolonged period of time, perhaps even for some time, because of a symmetrical weakness in the picture and at the loss of sensitivity. Nadal nabuvay has a progressive, recurrent or chronically monophasic character.
In a large number of cases, neurological symptoms do not occur, if there is an infection, it is necessary to clarify the relapsing history, especially the manifestation in relatives of stomatomy and weakness of the feet
z'yasuyut:
· Symptoms of polyneuropathy have long appeared (oniminnya shkiri, myazova weakness, etc.);
· Chi is not a ominous patient with alcohol;
· Chi tied yo professional efficiency with any kind of chemical words (gasoline, salts of important metals);
· Chi maє patsієnt cukrovy diabet (sickness, which is characterized by periodic or persistent glucose levels in the blood, which is toxicly infused on all organs and tissues);
· There were no similar symptoms in any of the closest relatives.
Zakvoryuvannya vinikє in any vіtsі, including a child. The HVDP is able to move the ear with the movers and the gradual development of symmetric weakness in the proximal and distal sides of the lower edges and paresis, with an extension of 8 types.

physical condition:
State-of-the-art status: foreign camp and severity, temperature of the body, vimiruvannya of the patient's vagi, inspecting the pupils' curves, behavior, pulse, AT, camp of internal organs (legends, heart, furnace, nirki and inn.
Neurological status: level of svidomosty, general cerebral symptoms, meningeal signs, cranial nerves, rukovo-reflex sphere, sensitive sphere, coordinator function, pelvic organ function, cognitive function of the nervous system, autonomic.
Paresis is usually overwhelmed in the legs, which is difficult to manifest when walking on gatherings, when standing up and down and falling. Weakness can be promoted in the proximal as well as in the distal muscles. The swelling of paresis is not supervised by an increase in atrophy; Characteristic of diffuse hypotonia of myaziv. Variations of the bolovy syndrome occur infrequently. Early and characteristic of CIDP є lowering of tendon reflexes in later visualizations. Naybіlsh permanently transmits the Achilles reflex.
Some of the ailments suffer from cranial nerves (call face and bulbar, older than okorukhs). Cranial neuropathy depends on the activity of CIDP. M'yazova weakness in supernatant sensory impairment in patients with hyperesthesia or anesthesia. Sensitive impairment (paresthesia, hyperesthesia, hyperpathy, hyperalgesia of the type "shkarpetok", "mitten") can also be tolerated in case of acute illness, and even if it does not appear in the clientele on the first plan.
If the brain is missing, it is possible to develop clinical signs of the central nervous system - the Babinsky reflex or the development of tendon reflexes. Vegetative impairment (overgrowth and acrophysihydrosis, passing of heart arrhythmia with development of obscurity, progressively decreasing the weight of the body by 10-20 kg in length at normal temperatures, during the duration of the cerebral and intestinal dysfunction) Dichally lack of development in CIDP is very rare.

In the classroom practice for assessing the degree of rotation of ruddy rosters, the scale of the strength of the varieties of colors (A. Szobor, 1976).

... 0 points - to ruku in myazuvidsutnі.

... 1 point - minimal ruku in music, the patient will not let go of the picture.

... 2 bali - the patient will utrim the vagu of the kintsevki, ale the opir, which is to be repaired to the glimpse, is minimal.

... 3 bali - patsієnt to repair the opіr to the Zusillians, the camp of the kinsіvki, albeit insignificant.

... 4 bali - patsієnt to kindly repair the Zusillas for the improvement of the camp of the kintzivka, ale and deyake of the decrease in strength.

5 points - the strength of the sound of the change and constitutional norms of the situation.

· More importantly, distal or changes (distal and proximal) nature of paresis of the teeth;
· Myazova hypotonia and hypotrophy of hands and nig are swirled;
· More parts and styles of sensitive and vegetative breakdown in pictures;
· Vkray rydko - bringing in spontaneous and pelvic dislocations.

The KhVDP is diagnosed with the presence of two regional key signs:
-progressive or recurrent weakness in the triviality of 6 months;
-hypo- abo areflexia in all chotiroh kintzivkah.

Diagnostic criteria of CIDP (A.Ropper, E . Wijdicks, V.Truax, 1991)
... bilaterally symmetrical weakness of the colors.
... paresthesia in the fingers of the fingers and nig.
... Progression from 6 days to 6 months, from 6 days to 6 months from 6 months to
... tendinous hyporeflexia in kintsivkah, denote the muscular weakness; visibility of the achilles reflexes.

Laboratory Doslidzhennya:
UAC - for the prevention of ignition of internal organs, for the prevention of polyneuropathic syndrome;
· Prescribed blood for tsukor, creatinin, sechovina, AST, ALT, bilirubin, on elektrolity - biochemistry of pretreatment of blood in addition to assisting in the inclusion of metabolic polyneuropathies;
· ОАМ - for vyklyuchennya zakvoryuvannya internal organs, for supervising polyneuropathic syndrome;
· Pre-treatment of blood for thyroid hormone - і thyrotoxicosis, і hypothyroidism may suffer from weakness, polyneuropathic syndrome;
· Pre-treatment of blood on VKV (value of antinuclear antibodies) - the KVDP clique has been pummeled with systemic ailments;
· PLR of blood for hepatitis virus - for the development of polyneuropathic syndrome in hepatitis;
· Pre-treatment of blood for VIL-infection - for the prevention of polyneuropathy, associated with VIL-infection;
· Immunelectrophoresis of blood cells - pretreatment of blood for blood cells, blood cells, for monoclonal proteins - for the prevention of disproteinemic polyneuropathy;
· Immunelectrophoresis of monoclonal proteins, BensJones' blocks allows the inclusion of monoclonal gammapathy and malignant ailments.

Instrumental doslіgennya:
· Oglyadova R-graph of the organs of the breast cancer - for the prevention of adolescent tuberculous infections, oncological diseases of the legends, in case of any possible development of polyneuropathic syndrome;
· Ultrasonic examination of organs of the worm-empty - the incidence of internal organs (liver, nirok and іn) can be supervised by polyneuropathy, similar to HVDP;
· MRI - to the brain - for the activation of the demilinization process in the central nervous system (detection of the demilinization in the cerebral stovbur, periventricular, subcortical);
· Ultrasound scan - thyroid zone - for carrying out differential diagnosis of CIDP with metabolic polyneuropathy;
X-ray of flat cysts - to carry out when paraproteinemia is detected for malaria disease
· Electroneuromyography (ENMG) * - allowing signs of destruction of muscle and axonal degeneration to appear.
· Holly elektromyografiya ** - by means of such є to reveal signs of a flow denervation-reinnervational process in polyneuropathy.

NB!* In case of systemic lesions of peripheral nerves, it is necessary to clarify the type of pathological process (axonal or demilinizing).
The main criteria for the axonal process:
· Decreased amplitude M - vidpovidі;
· Normal or slightly decreased performance of stimulation along motor and sensory axons of peripheral nerves;
· Detection of blocks, about how to lead a breakdown;
· Increasing the amplitude of the F-hwil, the appearance of the great F-hwils with the amplitude, scho changing 5% of the M-hwil amplitudes.
The main criteria for the demyelinizing process:
· Reducing the performance of stimulation on motor and sensory axons of peripheral nerves (on the hands less than 50 m / s, on the legs less than 40 m / s);
· Increase of triviality and polyphase M - changes;
· Increased residual latency (more than 2.5-3 m / s);
· Necessity of blocks of carrying out zbudzhennya;
· Expanded to the latency range of F-hvil.

During HVDP on the EMG, there are changes in the potentials of ruddy ones, which are characteristic of denervation, and a rapid stage of the development of fibrillation, so that it can be caused by triviality and severity of damage. The speed of carrying out on the loose and sensory fibers on the upper and lower edges will help to increase the decrease by 20% (as the demolition process is not surrounded by only the roots of the spinal nerves). There can be a change in the degree of rotation, blocks of carrying out and the time dispersion of the total muscular potential for the potential of the nerve fibers. Distal latencies in the case of a widespread ill health.

NB!** Until the most frequent distal joints of the upper and lower joints (for example, the anterior tibial joint, the back of the fingers), and, if necessary, the proximal joints (for example, the chotirigol). It is necessary to remember that the first signs of denervation to the process do not appear earlier than after 2-3 days after a cob of illness, but signs of the renewed process - not earlier than after 4-6 days. The appearance of the flow denervation-reinnervational process of additional assistance in the differential diagnostics of the HVDP and MGB in the spinal vypadki.

Diagnostic algorithm:

Diagnostic panel, proponed OK. Koski (L.C. Koski, 2009) including clinical and neurophysiological diagnostic criteria for CIDP.
Key criteria include:
- a symmetrical ear or a symmetry of an exercise in case of an active reading,
- the manifestation of weakness in some pictures and the manifestation of proximal weakness is not less, not in one picture.

Neurophysiological criterion includes the fact that there is a desire to be one of the signs of demilinization (in the reduction of the distal latency, for the decrease in the SRV, or the increase in the latency of the F-sickness) not less than in the 50% for the reduction of the nerves nerves). Assigned diagnostic criteria can be quite sensible and specific (more than 85%) and handy for practical storage.

Diagnostics (stationary)

DIAGNOSTICS at the stationary РІВНІ

Diagnostic criteria at the stationary level:
The classical CVDP is characterized by a symmetric weakness of the proximal and distal joints of the joints, progressing more than 2 months. Vona also associates with sensitive injuries, weakened or deficiencies of tendon reflexes, changes in the size of a child in spinal cord disease (SMR), changes, typical for demineralization of diseases This current can be recurrent or uninterruptedly progressive, and the remaining is more typical for young patients.

Diagnostic Criteria of the CIDP Working Group on the CIDP of the European Federation of Neurological Associations/ European Partnership for Implantation of Peripheral Nervous System (EFNS / EPNS) 2010 р include three blocks:
· Clinical criteria;
· Neurophysiological criteria;
· View signs.

The block of key criteria for transferring the conversion to the type of the key characteristics of the CIDP, which is considered to be one of the variants

Criteria for determining the diagnosis of CIDP

Switching of the main diagnostic entries:

· Analysis of the spinal cord;
· Electroneuromyography.

Change of additional diagnostic entries:
· Analysis of liquor for immunoglobulin;
· Holchasta elektromyografiya;
· MRI of nerve gossip;
· Biopsy of the lithic nerve.

Differential diagnosis

Likuvannya beyond the cordon

Pass a lykuvannya in Korea, Izrail, Nimechchin, USA

Otrimati the consultation on medturism

lykuvannya

Preparations (such as speech), which can become stagnant during treatment

A group of drugs in the absence of ATC, which can become stagnant during drug use

Likuvannya (outpatient clinic)

LIKUVANNYA ON AMBULATORNY RIVNI

Likuvannya tactics: go in, which is carried out with HVDP, on specific (pathogenetic) and nonspecific (symptomatic).
The plan for the treatment of the HVDP at the outpatient hospital has three main directions:
· Zupiniti autoimmune firing and ruining nerves;
· Renewal of nerve conductivity, force of communication and sensitivity (remієlіnіzatsії);
· Unique repetition of HVDP attacks.

non-druglykuvannya:
· Prevention of the regime of practice and correction (physical and psychological treatment can be provoked);
· Prototype proofs of the insolation, let it be a kind of infectious disease, cut it off, rob it, be it chippings and a test of Mantoux.

Drug treatment:
· In a given hour, there are three main pathogenetic diseases of CIDP-corticosteroids, plasmapheresis, immunoglobulin.
· Posindromic therapy.
Symptomatic therapy for CIDP includes:
· Preparations for the relief of neuropathic pain (gabapentin 1200-1800 mg / day, amitriptylin 75 mg / dobu);
· Anticholinesterase drugs (neostigmin 0.5 mg / dobu).

Algorithm for non-emergency situations:
· Symptomatic therapy

Інші vidi lіkuvannya:
· Plasmapheresis;
· Golkoreflexotherapy;
· Physiotherapeutic treatment;
· Physical culture;
· Hyperbaric oxygenation;
· Orthopedic come in with peripheral paresis.

Consultation of an infectious disease specialist - establishment of any chronic infection (brucellosis, boreliasis, etc.)
Consultation of a therapist - establishment of abnormal therapeutic incapacitation (fiery incapacitation of internal organs: legends, nirki, pechinka, etc.)
Consultation of an oncologist - if necessary, oncology
Consultation of an endocrinologist, nephrologist, rheumatologist - if necessary, the inclusion of somatic pathology

Prophylactic come in:
Primary prophylaxis: it is aimed at prophylaxis of zagostren.
· Post-treatment and regular examination by a neurologist є obovasovy for ailing CIDP.
deposited:
· Control immunograms once a day.
Not recommended:
· Ailments with infectious diseases, illnesses, which are transmitted by a state road;
· Zalishati without respect zagostrennya chronic ailments (tonsillitis, pharyngitis, sinusitis, pyelonephritis, gastritis, etc.).
· It is easy to put up to odyagu in cold weather, get cold;
· Piddavatisya be-any kind of bran (great doses of alcohol, contact with toxic farbs and varnishes, etc.);
· Launch the empty company (karієs, stomatitis and іn);
· Robiti be-like chips and Mantoux test;
· Visnazhuvatisya and overturned physically and psychologically;
Perebuvati pіd dієu active sleepy exchanges

Secondary prevention is focused on accelerated prevention:
With CIDP, spontaneous remissions are not susceptible, but for the prevention of recurrences, it takes a trivial hour to take immunosuppressive ones.
It is trivial to use glucocorticoids to produce side effects, such as Cushing's syndrome, osteoporosis, steroid diabetes, arterial hypertension, tendency to the growth of vagi, superficial gastritis.
· The tendency to the growth of vagi vimagає systematic vikorystannya otmezheniya kalorіnostі vіzhivanoї їzhі, especially in carbohydrates.
· The trend towards a decrease in tolerance to carbohydrates and a periodical increase of sugar in the blood, in the form of drugs prescribed, to increase the tolerance to carbohydrates, normalization of peroxidation before oxidation
· For the prevention of gastritis vikoristovuyu change the course of treatment with the proton pump with the ingibitor (omeprazole 20 mg 1 time for 10 days).
In case of sympathicotonia (adduction of an arterial vise, tachycardia, and even to prednisone psychosis), it is necessary to prescribe vegetotropic drugs (anaprilin 20-40 mg 2-3 times as needed). Most often it occurs during pulse therapy with methylprednizolone.

Monitoring the patient's stance- on this stage, according to the rules for maintaining medical documentation, establish (PMSD, medical centers, etc.), in which the following are re-instituted:
· Assessment of the ailing camp with a description of the camp of shkirnykh pokriviv; vagi of the sick;
· Indicators of hemodynamics: the number of spontaneous rucks, A / D, HR, Pulse;
· Assessment of neurological status.

· The visibility of the ignition of the ignition demilinizing polyneuropathy.

Likuvannya (shvidka dopoga)

DIAGNOSTICS І LIKUVANNIA AT STAPI SHVIDKOЇ NEVIDKLADNOЇ HELP

Diagnostics come in: depending on the state of the art, there is no need to help.

Drug treatment:
Ethiopathogenetic treatment is not carried out.
On this stage, symptomatic therapy is carried out.

Likuvannya (stationary)

LIKUVANNYA at the stationary РІВНІ

tactics of addiction
Non-drug treatment:
· Dyeta Nizkosolova with a high vm_lka, ale low vm_ fat;
· Zabezpechennya adequate to look for ailments with the method of prevention of bedsores and contractures.

Drug treatment:
Lіkuvannya CIDP is composed of pathogenetic and symptomatic therapy. Pathogenetic therapy is based on the development of immunomodulatory diseases, as in this day it was taken on the basis of the first and other lines.
In yakosti therapy of the first line vikoristovuyu:
Glucocorticoids (GC),
Human normal immunoglobulin for internal introduction (VVIG)
· Plasmapheresis.

HA (methylprednizolone or prednizolone)... For all ailments, it is recommended to take prenizolone in a dose of 1.5 mg / kg a day for 2-4 days, when polishing, I will step by step to take a dose of prenizolone every other day. In case of a significant excess of the period, the dose of prednisolone can be changed step by step (by 5 mg of skin 2). After 3-4 months, switch to an additional dose (20 mg every other day), and take more for 2 months. In such a rank, the course of treatment is trivial in the middle of 6 months. The prenizolone is skewed in case of repeated renewal of the potency of the principles and the pre-requisite indicators of the electro-neuromographic (ENMG) for the last time.
Alternative treatment schemes: methylprednizolone intravenously 1 g / day 5 days, sometimes 1 g / add 1 time per day for 4 or 8 days, use 1 time per month; or 500 mg of methylprednizolone orally 1 time per week 3 m / s, then the dose should be reduced by 50 mg of skin 3 m / s.

plasmapheresis
In the given hour, plasmapheresis as monotherapy is stagnant in CIDP in vinyatka vines: resistance is either contraindicated to the persistence of corticosteroids and / or immunoglobulin in class G.
Then call up plasmapheresis with CIDP to go to hormone therapy (Div. Vishche). In a wide range of operations, up to 200-220 ml of plasmas / kg are carried out per course for 5-7 sessions with two lengths. As a substitute component in plasmapheresis, it becomes 5% of albumin levels, so as the drug, on the basis of fresh frozen plasmas, gives a lesser number of accelerated and replenishment of the rhizik of the transferred hepatitis B and CHIDy.
In quiet patients, if plasmapheresis is carried out with CVDP as monotherapy, it is recommended that it be taken 2 to 3 times per week before the appearance of a stable clinical course with further changes in the frequency of operations with a stretch of deciduous months.

In the last few days, it was shown before plasmapheresis with CIDP, they knew a lot of evolu- for some reason, it is not a matter of consuming immunoglobulin in class G).
Plasmapheresis without oven for vaginal women and children, who are suffering from the HVDP. It is not recommended to use plasmapheresis with corticosteroids, as it will reduce its therapeutic efficacy. Approximately 5-10% of the ailments who suffer from plasmapheresis are susceptible to relapse and relapse, which, as a rule, quickly regress to the smallest morbidity patterns.
An alternative to plasmapheresis is intravenous immunoglobulin (IVIG).
VVIG zazvichayut in a nasal dose of 2 g / kg (stretching 2-5 dB.), Poten 1 g / kg skin 3 tension. stretching 6 ms. or 0.5 g / kg for a stretch of 4 days for leather months for a stretch of 6 ms.

The therapeutic effect of immunoglobulin is based on anti-firing, neutralization of anti-virus, antibacterial antibodies and autoantitils, anti-inflammatory and neutralization of active complement. For the effectiveness of immunoglobulin before plasmapheresis, ale less acceleration (head bile, shkirnі reactions, bіl across, chills, aseptic menіngіt). Immunoglobulin of contraindications We are sick with lack of immunoglobulin A, as they have a risk of development of anaphylactic shock.
If there is a lack of efficiency in the first line, the reserve drugs are stored - pick up another line- azathioprine, cyclophosphamide, cyclosporine, mycophenolate mofetil, methotrexate.

Azatioprin (imuran) prescribe orally at a dose of 50 mg / cyt with increased doses of up to 150-200 mg / cyt.

cyclophosphamide vicorisation during the daytime as a result of the other immunodepressants as in monotherapy, as well as in conjunction with azathioprine. The drug is administered internally at a dose of 200 mg or every other day at a dose of 400 mg. Therapy can be repaired in the minds of the hospital, and only after having passed into the tolerance of the drug, it is possible to transfer the ailments to the outpatient clinic. cyclosporine it is prescribed orally at a dose of 3 mg / kg. When there are toxic reactions, the dose can be increased up to 5 mg / kg 2 times a day. To achieve a stable therapeutic effect, the dose of cyclosporine can be reduced to a minimum

mycophenolate mofetil prescribe a supplement to glucocorticoids in vipads, if a lower dose is required. Mycophenolate mofetil should be administered orally at a dose of 2 g / cyt with a similar correct dose, depending on the efficiency of treatment.

tіamіn- priznachayut 25-50mg 2-4 times for doba. Course 3-4 days.

The change of the main lykarsky zoobіv:

medical rehabilitation

carried out according to the Standard of Organizational Assurance of Medical Rehabilitation to the Population of the Republic of Kazakhstan, approved by the order of the Ministry of Health Protection of the Republic of Kazakhstan dated 27.12.2013, Rock No. 759.

palliative assistance

On this stage, the psychological education of the sick and relatives, legal and social nutrition is carried out on the basis of the improvement of the quality of the life of the sick and the motherland.

Falling down to the type and severity slowed down, but ailments have come to a head, you can also know more about it, like:
For those who are sick, we have to admit heparin for children in a dose of 5,000 OD for skin 12 years and an hourly compression of lithic mucous membranes to prevent glib vein thrombosis
· Massage is beneficially infused into the exchange of speech, which will also accelerate the growth of nerves and renewal.
Kinesiotherapy brought stimulation to renewal and renewal of myazovy volume
· Physiotherapy for polishing strength, for preventing the formation of contractures (electrostimulation, heat treatment, lykarsky electrophoresis).
· Reabilitation for the development of common tips and adaptive products that add to everyday life.
A person who is sick can know orthopedic as well as additional additional methods of oversupply
Psychotherapy

hospitalization

Indicated for planned hospitalization:
· Zagostrennya KVDP;
· Increase in sedentary neurological symptoms in patients with chronic igneous demilinizing polyneuropathy.

Indicated for the emergency hospital:
· Patients with previous diagnoses of HVDP.

information

Dzherela and Literature

1. Protocols of the United Committee for the quality of medical services of the Ministry of Health and Social Development of the Republic of Kazakhstan, 2016
   1. 1. Bikova O. V., Boyko A. M., Maslova O. I. Internal stasis of immunoglobulins in neurology (Review of literary and vlast sparing) // Nevrol. zhurn. - 2000, 5.P.32-39. 2. Gekht BM, Merkulova DM Practical aspects of the clinical course and treatment of polyneuropathies // Nevrol. zhurn.-1997 No. 2.-P.4-9. 3. Mozolevsky Yu. V., Dubanova E. A., Ivanov M. І. Klinika and likuvannya of chronic ignition demilinizing polyradiculoneuropathy // Zhurn. neurol. і psychiatr. - 1992, No. 3. P.106-110. 4. Latov N. Advances in the diagnosis and treatment of CIDP and related immune-mediated neuropathies // Neurology. - 2002. - Vol. 59 (Suppl 6). - S. 1. 5. Akhmedova G.M., Averyanova L.A., Devlikamova F.I. that in. Complex dosage of chronic dizimmune neuropathies for lesions of the central nervous system // Practical medicine. - 2013. - T. 68, No. 1 (1). - S. 83-86. 6. Vallat J.M., Sommer C., Magy L. Chronic inflammatory demyelinating polyradiculoneuropathy: diagnostic and therapeutic challenges for a treatable condition // Lancet Neurol. - 2010. - Vol. 9. - P. 402-412. 7. Dyck P. J., Lais A. C., Ohta M. et al. Chronic inflammatory polyradiculoneuropathy // Mayo Clin Proc. - 1975. - Vol. 50. - P. 621-637. 8. Gorson K.C., Katz J. Chronic Inflammatory Demyelinating Polyneuropathy // Neurol Clin. - 2013. - Vol. 31. - P. 511-532. 9. Misra U.K., Kalita J., Yadav R.K. A Comparison of Clinically Atypical with Typical Chronic Inflammatory Demyelinating Polyradiculoneuropathy // Eur Neurol. - 2007. - V. 58. - P. 100-105. 10. Koller H., Kieseier B. C., Jander S. et al. Chronic Inflammatory Demyelinating Polyneuropathy // NEJM - 2005. - Vol. 352 (13). - P. 1343-1357. 11. De Sousa E.A. Chronic inflammatory demyelinating polyneuropathy: diagnosis and management // Expert Rev. Clin. Immunol. - 2010. - Vol. 6 (3). - P. 373-380. 12. Busby M., Donaghy M. Chronic dysimmune neuropathy: a subclassification based upon the clinical features of 102 patients // J Neurol. - 2003. - Vol. 250. - P. 714-724. 13. Van den Bergh P.Y.K., Hadden R. D. M., Bouche P. et al. EFNS / PNS Guideline on management of chronic inflammatory demyelinating polyradiculoneuropathy: Report of a joint task force of the EFNS and the PNS - First Revision // Eur J Neurol. - 2010. - Vol. 17. - P. 356-363. 14. Koski C.L., Baumgarten M., Magder L.S. et al. Derivation and validation of diagnostic criteria for chronic inflammatory demyelinating polyneuropathy // J Neurol Sci. - 2009. - Vol. 277. - P. 1-8. 15. Brannagan T.H. Intravenous gammaglobulin (IVIg) for treatment of CIDP and related immunemediated neuropathies // Neurology. - 2002. - Vol. 59 (Suppl 6). - S. 33-40.27. Koski C.L. Therapy of CIDP and related immunemediated neuropathies // Neurology. - 2002. - Vol. 59 (Suppl 6). - S. 22-27. 16. Rajabally Y. A., Narasimhan M., Chavada G. Electrophysiological predictors of steroid-responsiveness in CIDP // J Neurol. - 2008. - Vol. 255. - P. 936-938.

information

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One of the slowdowns in alcoholic beverages is alcoholic polyneuritis, which is a disease, with any deterioration in the function of peripheral nerves as a result of the infusion of toxicogenic metabolites into ethanol on the muscle membrane of the nerve cells. Most often polyneuropathy of the supernatant of the stage of alcoholism, with which the regular intake of alcohol is promoted. Ailments appear in 50-80% of chronic malignancies of alcoholic beverages. Naybilsh was sick to the disease of a man. The ICD-10 (international classification of ailments) is known to the given incapacitation code "G62.1".

It is consistent with the reduction of nerve cells, as well as the clinical picture of alcoholic polyneuropathy, which can be categorized as follows:

1. The sensory form is characterized by the expression of sensitive fibers. Symptom complex of representations of unacceptable visions, pains, decreased sensitivity of minds and shkiri of lower symptoms, deeper - hands.
2. Rukhov's form is motor, amazed by the deterioration of the function of the ruk's neurons, superfluous syndrome and painful symptoms, as well as diminished functions of loafs, ailments to the loss of laxity, homozygosity in the number As a result of a decrease in rukhovoy izdatnost і at a trivial interruption of the loss of life, the deterioration of the atrophy of the mutilation of the parts of the body.
3. Zmishana is the presence of signs of both sensory and motor polyneuropathy, such a form is a legacy of the protracted nature of the overcoming of the ailment and the presence of adequate drug therapy and physiotherapeutic visits.
4. Peripheral pseudo-obesity of alcoholic genesis (atactic form) for its manifestations is similar to tabes of the brain, which develops in syphilic lesions of the central nervous system, with a provincial symptom of impaired coordination.
5. Subclinical manifestation in a greater number of ailments with chronic alcoholism, in case of a lack of scarg on the side of a neurological status, the battle can only be attributed to additional diagnostics.
6. Vegetative polyneuropathy can lead to dysfunction of internal organs.

Possibly a few options for overcoming ailments:

• gostre;
• chronic;
• asymptomatic.

cause

I will increase the pathogenesis of alcoholic polyneuropathy to become unvaccinated. The main role in the development of ailments is played by three main officials:

• injecting ethyl alcohol and acetaldehyde metabolism on the lipid membranes of axons: there is a lack of blood circulation in the nerve cells;
• failure of vitamins of group B, especially thiamin (B1), as a result of insufficient alimentary care and alcohol consumption. In case of malignant alcoholic beverages, the moisture in the intestines breaks down, there is a lot of deficiency in vitamins, so there is also a great role in the development of avitaminoses, which is ruined by the mode of eating in ailments;
• Intensive establishment of radicals is harmful to the health of endothelial cells, as a result of the development of endothelial cells, as a result of which endoneural hypoxia develops.

symptoms

The development of polyneuritis is supervised:

• unacceptable viglyadi visions of goosebumps and pokolyuvan shkiri;
• a variety of different groups of languages ​​in case of atrophies, most often distal or lower symptoms, as a result of which the move is broken;
• by the ships of litkovy m'yazіv, scho to agree at night;
• a decrease in sensitivity in the lower views, especially the feet, as a result of which there can be traumatization with the development of secondary infectious processes, in which there is a similarity between alcoholic and diabetic polyneuropathy;
• m'yazovi more baking character;
• deterioration of rіvnovagi yak inheritance urazhennya kіtin mozochka (signs of ataxia);
• deterioration of microcirculation, at which there is a coldness of the feet and hands, and a symptom of a disease is caused by a breakdown of the vesicles of capillaries;
• vegetative impairment: non-tampering of sech, or navpak, as a result of the development of neurogenic sectoral malignancy, impaired erectile function, impaired intestinal motility, constipation, as a result of the development of neurogenic secular malfunction, impairment of intestinal motility, constipation, such as disgusting medication.

diagnostics

A person who is sick, who is suffering from alcoholic depletion, needs the watchfulness of a psychiatrist or a narcologist. When the first signs of polyneuropathy appear, they go to the neurologist for further diagnostics and a sign of the treatment plan.

The diagnosis of alcoholic polyneuritis is made according to the ICD at the office:

• anamnesis, in which there is a pathogenetic lanka in viglyadі overworldly implantation of alcoholic beverages;
• skarg for a decrease in the sensitivity of pictures, atrophies of m'yazov, deranged walking, inappropriate perceptions on shkiri, m'yazov pains and sudomi;
• physical look, at any time there are signs of sensory, motor and vegetative damage in the eyes of a decrease in reflexes, sensitiveness of shkiri, a decrease in the tone of muses, a reduction in looseness in the slopes;
• Instrumental views of the preliminaries: effective electro-imaging, as the electrical potential of the muscles appears, with neuropathy, there will be a decrease in patients with subclinical interruption of ailments;
• in some cases, a biopsy of the nerve is stagnated for a second histological analysis.

lykuvannya

Therapy of alcoholic polyneuropathy is based on a complex approach to the pathogenesis and elimination of symptoms.

1. Vidmova from the malignancy of alcohol is the main stage in the treatment of polyneuritis, vicious to the malignancy of ethyl alcohol. If a patient does not want a viscous croc, then the medication therapy can be lost without an effect.
2. Vitamin complex of vitamins of group B, especially tiaminu. It is used orally, as well as in the case of infusion therapy. In medical practice, the following drugs are most often used for the treatment: Neurorubinu, Trigamma.
3. For the training, I will become and update the structure of the muslin shells of neurons in the vikoristovuyu to be. scho to enter the pharmacological group natrapiv. Dozіlno zasosuvannya Phenibut, Cerebrolizinu, Tenoten.
4. Preparations, direct to microcirculation of blood - Tiolepta, Dialipon.
5. The relief of pain syndrome can be done with the help of non-steroidal anti-inflammatory drugs - Ibuprofen, Diclofenac. For protection, I have given a group of drugs to treat the ailments, so we suffer from chronic ailments of the intestinal tract, especially the inferior ailments, as in the case of speech. go to the warehouse of medicines to stimulate the establishment of the mucous membranes of the etching organs.
6. Dietotherapy is based on respect for the correction of the harvesting regime: the reception of the person is guilty of shot and frequent, and all the necessary vitamins and microelements are included in the store of guilt.
7. Sedative and spoky in a way є poultry, mint chamomile, tea based on these can be helpful in the period from which to drink and from alcoholic beverages.
8. Physiotherapy for the renewal of the absorbed amplitude of the rucks in the slopes.

Your Narcologist is in advance:

Neuropathy can lead to development without delay:

• the deterioration of the function of the stove;
• heart and nirkova lack;
• alcoholic encephalopathy;
• erectile dysfunction;
• ruinous sechovipuskannya;
• lesion of the corns;
• parallel lower and upper patterns;
• Decrease in the state property for the zoru.

prophylaxis

The main approach of prevention is the diagnosis of polyneuritis of alcoholic genesis є vidmov by implantation of drinks on the basis of ethyl alcohol. In order to meet the needs of the immune system, it is necessary to maintain the correct way of life:

• active sports;
• walks on a fresh occasion;
• vidmova vid tyutyunopalinnya.

In order to achieve success in the fight against fallowness, it is not necessary to receive advice from a psychologist and to be careful with a psychiatrist, since the therapy of chronic alcoholism includes both the correction of the psychotic therapeutic status, and the trivialization of complex medical treatment.

forecast

Nybilsh subclinical and mild forms of alcoholic neuropathy. With a free diagnostics and early attribution of an elusive drug, it is possible to renew the structure of the nerve sheaths. A protracted recession of ailments and it is important to form a form before the invalidization and acceleration, as it is disgusting to indulge in a liking.

The Russian International Classification of ailments of the 10th glance (ICD-10) has adopted a single normative document for the incidence of illness, the reasons for the infestation of the population to the medical establishments, the causes of death.

ICD-10 was introduced into the practice of protecting health in the entire territory of the Russian Federation in 1999 by the order of the Ministry of Health of Ukraine dated 27.05.97. No. 170

A new look (ICD-11) is planned by WHO in 2017 2018 roci.

Іf changes and additions of the VOOZ rr.

Processing and transfer zmin © mkb-10.com

Diabetic polyneuropathy code according to ICD-10

Polyneuropathy is a complex of ailments, which includes multiple lesions of peripheral nerves. It is most often necessary to move into the chronic stage and by the volodya with the lower path wider, so that the process of collecting the extra fibers and step by step is going to take all the big bugs.

Classification of polyneuropathy according to ICD 10 є officially known, but not for individual specific features, and does not describe the tactics of treatment.

Symptoms and diagnostics

A clever picture in the first place to be grounded on the ruined robots of the support-and-meat apparatus and the heart-vascular system. Patient skarzhitsya on pain in the muscles, their weakness, judgment and the appearance of the building to normal oversaturation (paresis of the lower symptoms). Frequent frequency of the heart rhythm (tachycardia), symptoms of arterial grip, hypothermia and headaches through changes in the tone of the brain and improper blood circulation of the organs of the central nervous system are given to the backward symptomatology.

When the health of the ailing ointment is gone, I will atrophy, the lyudin basically lie, but it is negatively signified on the grating of the soft tissues. In some cases, necrosis develops.

A handful of remedies for goiters to listen to all of the patient's skargi, to take a look around, to reconsider the tendon reflexes and the sensitivity of the pupils for the help of special tools.

Laboratory diagnostics of blood is effective in terms of the presence of specific pathologies and the reasons for the development of the main disease. It is also possible to increase the adjustment of glucose concentration in non-toxic spores, salts of important metals.

І of the modern instrumental methods for the reduction of electrical neuromography and biopsies of the nerve.

lykuvannya

The International Committee has developed a system for the treatment of polyneuropathy. In the first place, there is an infusion of the head causative factor - to become feigned by organisms for the help of antibiotics, to compensate for the impairment of the endocrine system and hormonal therapy, to change the mind of robots, to change the form of being involved in new alcohol intake, to become operative.

In order to unify the development of an accelerated development, a high-calorific value of the day (at a time of day), a complex of vitamins and minerals, which introduce the robot of the immune system and the trophism of a cell, is indicated.

For the knowledge of symptoms, there are no painful drugs, antihypertensive drugs, as well as muscle stimulants.

Treatment of diabetic polyneuropathy

Succinic diabetes є we will broaden illnesses in the whole world. Most often, it is most common to develop chronic diseases. One of the accelerated disease is diabetic polyneuropathy.

Chronic diabetic (sensorimotor) polyneuropathy - the form of neuropathy is broadened, as it is super-susceptible to sensitive, vegetative and ruffled disorders.

E 10.42 diabetic polyneuropathy in type 1 diabetes,

E11.42 diabetic polyneuropathy in DM 2,

G 63.2 Diabetic distal polyneuropathy.

Diabetic polyneuropathy is associated with pain syndrome and often decreases the level of life of patients.

The development of a sickness can lead to more serious acceleration. Such, yak: ataxia, Charcot's angle, diabetic foot syndrome, diabetic osteoarthropathy.

Diabetic polyneuropathy of children can be brought to gangrene and other amputations.

To that, it is important to remember the development, and it is more effective to pick it up even with the first signs of a child with a baby with diabetes.

cause development

The main ethiological factors that provoke the development of diabetic polyneuropathy are taken into account:

1. Courinnya and alcohol;
2. Failure to control blood glucose;
3. vik;
4. Arterial vise;
5. Destruction of spіvvіdnoshennya lіpіdіv (fatty words) blood;
6. Decreases against insulin in blood;
7. Trivial overbig of a diabetic.

The number of up-to-date ones should be taken into account for those who constantly control the level of glucose and the arterial grip, but rather reduce the development of pathology. And the time spent on insulin therapy will reduce the risk of development by a factor of two.

symptoms

Symptoms of diabetic polyneuropathy appear as more symptoms in the lower symptoms. Pekucha, stupid or sverblyachі bіl, rіdsche gostry, prickly і pierced. The most frequent winners are in the foot and to feast on the evening hour. Nadal, more can be manifested in the lower third of the woman's hands.

Patients skarzhitsya on the part of the m'yaziv, pain in the slopes, the ruined walk. Tse is tied with a rosette in the nervous system. Temperamental sensitivity is involved, trophism may appear.

Hvory vidchuv discomfort comes from doty ogyagu. Pain syndrome in such vipadas is of a permanent character and sutta pogirshuє in the back of the self-esteem of the sick person.

Yak viyaviti and clarify the diagnosis?

Diagnosis of polyneuropathy is to be repaired from a doctor's account, which can help you collect anamnesis and a sign of the need for a diagnosis.

Electroneuromyography appears in the capacity of the main preliminaries. In addition, there can be stasosovuvati doslіdzhennya VKSP (vegetative shkіrnyh sympathetic potential).

lіkuvannya pathology

To set the diagnosis for diabetic polyneuropathy, use ethiotropic therapy. It is important to normalize glucose instead of blood glucose. For continuous control, in 70% of cases, more responses change. In some cases, insulinotherapy is indicated.

In the scheme of treatment for oxidative stress, for renewal of edema, it is necessary to use medicinal preparations with an antioxidant effect. By taking the drug, take courses to finish a trivial hour. At tsei period ide control and caution for the patient.

For pain relief, analgesics and protivopalny drugs are used. Ale, as I mean fakhivtsi, I will increase the stink of the odd odor from most of the seen, that trivially, you can get the correct robot into the slurry.

For symptoms of chronic neuropathic pain, anesthetics, antidepressants and antiepileptic drugs are prescribed. In addition to preparations, it is recommended to use vikoristovuvati plasters with lidocaine, gels, ointments and cream.

As a consolidation of a complex treatment of diabetic polyneuropathy, in the presence of a patient, I mean:

• fizio likuvannya,
• magneto and phototherapy,
• electropharesis i strumi,
• electrostimulation of myaziv,
• golkovkolyuvannya,
• hyperbaric oxygenation,
• monochromatic information optimization.

Likuvannya by folk zasoba is allowed to be deprived of a likuyuchiy lykar for the season. As an addition to the traditional methods of treatment, you can use phytherapy and treatment of ointments.

Effective treatment of diabetic polyneuropathy takes into account the individual's condition to the skin patient with a complex of conservative methods of treatment.

neuropathy

Code for MKH-10

name

to describe

The symptomatology of neuropathy is due to the nature of the nervous system and the growth of the brain. Most often, there are wineries in cases of foreign illnesses, early intoxications, and in some cases - in cases of early injuries.

The most common forms of neuropathy: diabetic, toxic and post-traumatic.

cause

Decrease of nerve throats in case of diabetic diabetic symptoms of growths of suckers and lipids in the blood, and cob lesions of many found vessels, how to liven up nerve fibers.

Post-traumatic neuropathy develops as a result of suppression and destruction of nerve fibers. It is not often the nerve conductivity that breaks down due to severe injuries, for example, a strong blow, which can cause the nerve sheaths to break down.

A major contribution to the development of neuropathy can be made by arthritis, nirkov and pechinka deficiency, hypothyroidism, swelling and malnutrition.

symptoms

Find the new form of neuropathy in the case of diabetes mellitus. When diabetic suffers, in the first place, the other children themselves, including those who supply blood to the nerve fibers. The main type of rice is the formation of neuropathy є a decrease in sensitivity in the areas of shock. In the past, the risk of traumatization and information on the incidence of severe problems in the area of ​​discomfort is growing. In case of diabetic, such a picture is found for the lower pictures.

There is a great part of patients with diagnoses of diabetes mellitus that they may have this form of neuropathy:

Peripheral: in general, in case of severe nerves, when they see for the innervations of the upper or lower symptoms, the visualization of those on the sides of the affected nerve; ailments mean a deterioration in the sensitivity of the fingers of both hands, as well as seeing them.

Proximal: the deterioration of sensitivity in the area of ​​homily, stegon and child is meant.

Autonomous: the activity of the organs of etching, the secular vision of any other organs will be destroyed.

The abnormal weakness in meat is also often a supernumerary form of diabetic neuropathy. When a lot of m'yazi stepwise atrophy, destructive curves develop.

Clinic of toxic neuropathies.

The reason for the given form of becoming ill is of the kind of intoxication. Nerve dissection can be prevented in case of infectious diseases (diphtheria, VIL, herpetic infection), as well as in case of disfigured speech (alcohol, lead, mischief), as well as in case of wrong

Alcoholic neuropathy is important for the development of the peripheral nervous system, as well as for the most frequent acceleration of non-alcoholic implantation of alcohol and surrogates. Symptomatic forms of alcoholic neuropathy appear practically in everyone to be ill with alcohol.

At the present moment, it seems that the infusion of the most outworn speech on the nerve fiber, and the destruction of the communication processes in the way of disabling the organisms, take the development of neuropathy.

More often the nerves of the pictures are suffering. Necessarily from the causes of rejection, neuropathy manifests itself in a deterioration of sensitivity in the feet and hands, the appearance of a feeling of feeling and poking on the skirts, hyperemic schools of children. Likewise, at larger stages of incapacitation, there may be a thickening of fabrics in the lower cues. Tsia Khvoroba is a protracted trip, the need for prophylactic visits, zokrem, sanatorium-and-spa treatment.

Clinic of post-traumatic neuropathy.

The reason for the post-traumatic lesion of nerve fibers is the delay in the result of fractures, tissue filling, improper coverage of post-traumatic scars, and new diseases. More frequent forms of this disease are lesions of the lytic, sinus and exchange nerve. At the same time, atrophy of muscular tissue develops, the destruction of fast-feeling health and a decrease in reflexes. It also implies a decrease in sensitivity to more podraznikiv.

lykuvannya

In case of toxic damage, it is necessary to pinch the toxic injection (skasuvati lykarsky drug, viklyuchiti priyom otruynykh rhechovin). Lіkuvannya diabetic form of getting sick, first for everything, until the normal level of blood sugar is obtained. In case of post-traumatic lesions of nerve fibers, it is necessary to use the optimal way to get rid of the inheritance of the traumatic factor.

Regardless of the form of sickness, the ailment is recognized as painful drugs, special groups of vitamins, and other drugs that will diminish communication processes and stimulate regeneration. Physiotherapeutic treatment is carried out according to the doctor's qualifications.

An important role is played by the prevention of neuropathies. It is brought up to the normalization of communication processes, a clockwise treatment of systemic and infectious diseases, as well as a quick stimulation of the mucous membranes in the course of orthopedic treatment.

I will look at those who are given to become ill often turn into a chronic form, it is necessary to live all the visits to get stung. For many ailments with neuropathies, go to the sanatorium-resort lykuvannya. In sanatoriums for the treatment of neuropathy, the following procedures are stagnant:

Exercise therapy and massage with acupuncture treatments;

Before the hour of the sanatorium-resort treatment, we are also averse to recommend therapy with a large number of vitamins in groups B, C and E. At the same time, it’s a great memory of being in the sanatoriums in order to get the chronicle of the process of taking over the time.

ICD 10. Class VI (G50-G99)

ICD 10. Class VI. Ailments of the nervous system (G50-G99)

EMPLOYMENT OF OKREEM NERVES, NERVOUS roots and gossip (G50-G59)

G50-G59 Management of nerves, nerve roots and gossip

G60-G64 Polyneuropathies and lesions of the peripheral nervous system

G70-G73 Ailments of the nerve-muscle synapse and muscle

G80-G83 Cerebral palsy and other parallel syndromes

The third category is as follows:

G55 * suppression of nerve roots and gossip with ailments, classified headings

G73 * Improvement of the nerve-muscle synapse and muscle in case of ailments, classified in the same headings

G94 * Іnshi lesion of the brain with ailments, classified in the same headings

G99 * Іnshi lesion of the nervous system with ailments, classified in the other headings

Viklyucheni: current traumatic lesion of nerves, nerve roots

і gossip - see nerve injuries in body areas

G50 Tricular nerve lesion

Includes: lesion of the 5th cranial nerve

G50.0 Neuralgia of the tricular nerve. Syndrome of paroxysmal facial pain, painful tic

G50.1 Atypova lice bill

G50.8 Inshi lesion of the tricuspid nerve

G50.9 Lesion of tricular nerve, unspecified

G51 Facial nerve lesion

Includes: lesion of the 7th cranial nerve

G51.0 Parallich Bell. Special parallich

G51.1 Firing of the university colincy

Excluded: postherpetic firing of the university colincy (B02.2)

G51.2 Rossolimo-Melkersson syndrome Rossolimo-Melkersson-Rosenthal syndrome

G51.3 clonic hemifacal spasm

G51.8 Lesion of facial nerve

G51.9 Lesion of facial nerve, unspecified

G52 Lesion of cranial nerves

G52.0 Striking of the nasal nerve. 1st cranial nerve dissection

G52.1 Cutting of the ulopharyngeal nerve Stripping of the 9th cranial nerve. ulicoopharyngeal neuralgia

G52.2 Cutting of the bloating nerve Pneumogastric (10th) nerve dissection

G52.3 Striking of the pid'yasic nerve. 12th cranial nerve dissection

G52.7 Multiple lesions of the cranial nerves. Polyneuritis of the cranial nerves

G52.8 Impairments of other refined cranial nerves

G52.9 Cranial nerve lesion, unspecified

G53 * Exercise of cranial nerves with ailments, classified in the same headings

Firing the ganglion of the university colincya

Neuralgia of the tricular nerve

G53.2 * Multiple lesions of cranial nerves in sarcoidosis (D86.8 +)

G53.3 * Multiple lesions of the cranial nerves in neoplasms (C00-D48 +)

G53.8 * Іnshi lesion of cranial nerves with other ailments, classified in the other headings

G54 Training of nerve roots and gossip

Viklyucheni: current traumatic lesion of nerve roots and gossip - divas nerve injury in the areas of the body

neuralgia abo neuritis NOS (M79.2)

neuritis abo radiculitis:

G54.0 Shoulder gossip cuts. Infratoracic syndrome

G54.1 Striking cross-crooked gossip

G54.2 Cutting out of the cores, not classified in the same headings

G54.3 Breast cuts, not classified in other headings

G54.4 Cutting of the cross-crooked roots, not classifying the bath in the same headings

G54.5 Neural amyotrophy. Parsonage-Aldren-Turner syndrome. Shoulder operative neuritis

G54.6 Kintzivka phantom syndrome with pain

G54.7 Kintzivka phantom syndrome without pain. Kintzivka phantom syndrome NOS

G54.8 Inspection of nerve roots and gossip

G54.9 Lesion of nerve roots and gossip, unspecified

G55 * suppression of nerve roots and gossip with ailments, classified in the same headings

G55.0 * suppression of nerve roots and gossips in neoplasms (C00-D48 +)

G55.1 * suppression of nerve roots and gossips in cases of fracture of the mid-spine discs (M50-M51 +)

G55.2 * suppression of nerve roots and gossips in spondylosis (M47. - +)

G55.8 * suppression of nerve roots and gossips in case of other more cinnamon, classified in the same headings

G56 Mononeuropathy of the upper picture

G56.0 Carpal tunnel syndrome

G56.1 Induction of median nerve

G56.2 Lactic nerve lesion Pizniy paralicheskogo nerve

G56.3 Exercise of the exchange nerve

G56.8 Іnshі mononeuropathies of the upper kіntsіvka. Interdigital neuroma of the upper bone

G56.9 Mononeuropathy of the upper kidney, unspecified

G57 Mononeuropathy of the lower picture

Excluded: more traumatic injury of nerves - see injury of nerves in areas of the body

G57.0 Lesion of the sciatic nerve

Dressings for lesions of the mid-spine disc (M51.1)

G57.1 meralgia paresthetic. Stem nerve syndrome

G57.2 Cutting of the stygular nerve

G57.3 Lesion of the bicular pidcolic nerve Parallich of small (Peroneal) nerve

G57.4 Lesion of median pidcolic nerve

G57.5 Syndrome of tarsal canal

G57.6 Pediatric nerve lesion Morton's metatarsalgia

G57.8 Interdigital neuroma of the lower bone

G57.9 Mononeuropathy of the lower brain, unspecified

G58 Іnshі mononeuropathy

G58.0 Intercostal neuropathy

G58.7 Multiple mononeuritis

G58.8 Other types of mononeuropathy

G58.9 Mononeuropathy, unspecified

G59 * Mononeuropathy with ailments, classified in the same headings

G59.0 * Diabetic mononeuropathy (E10-E14 + with the fourth digit 4)

G59.8 * Іnshi mononeuropathies with ailments, classified in the other headings

Polyneuropathies TA ІNSHІ URAZHNYA of the peripheral nervous system (G60-G64)

Viklyucheni: neuralgia NOS (M79.2)

peripheral neuritis with vaginosis (O26.8)

G60 Spadkov and idiopathic neuropathy

G60.0 Spadkov motor and sensory neuropathy

Spadkov motor and sensory neuropathy, type I-IY. Hypertrophic neuropathy in children

Peroneal muscle atrophy (axonal type) (hypertrophic type). Russi-Levi syndrome

G60.2 neuropathy in recurrent attacks

G60.3 Idiopathic progressive neuropathy

G60.8 Secondary depression and idiopathic neuropathies. Khvorob Morvan. Nelaton's syndrome

G60.9 Spadkova and idiopathic neuropathy, unspecified

G61 Ignition polyneuropathy

G61.0 Guin-Barré syndrome Hostry (post-) infectious polyneuritis

G61.1 Syrovatkova neuropathy. If it is necessary to identify the cause of the vikoristovuyut additional code of the underlying reasons (class XX).

G61.8 Інші ignition polyneuropathies

G61.9 Ignition polyneuropathy, unspecified

G62 Inshi polyneuropathies

G62.0 Lykarska polyneuropathy

G62.1 Alcoholic polyneuropathy

G62.2 Polyneuropathy, viclicanum and other toxic speech

G62.8 Inspection of polyneuropathies. exchange polyneuropathy

If it is necessary to identify the cause of the vikoristovuyut additional code of the underlying reasons (class XX).

G62.9 Polyneuropathy, unspecified neuropathy NOS

G63 * Polyneuropathy with ailments, classified in the same headings

G63.2 * Diabetic polyneuropathy (E10-E14 + with the fourth digit 4)

G63.5 * polyneuropathy in systemic lesions of the full tissue (M30-M35 +)

G63.8 * polyneuropathy with other ailments, classified in the same headings. Uremic neuropathy (N18.8 +)

G64 Іnshі rosters of the peripheral nervous system

Rozlad of the peripheral nervous system and NOS

Sickness of nerve-muscle synapses І М'ЯЗІВ (G70-G73)

G70 Myasthenia gravis and loss of nerve-muscle synapse

transient neonatal Myasthenia gravis (P94.0)

Yakshcho ailment wiklikan likarskiy zasoba, for its identification the vicoristovyut additional code of particular reasons

G70.1 Toxic damage to the nerve-muscle synapse

If necessary, identify a toxic word for a specific reason (class XX).

G70.2 Vrodzhena abo nabuta mistenia

G70.8 Disorders of the nerve-muscle synapse

G70.9 Loss of nerve synapse, unspecified

G71 First battle

Symptoms: multiple congenital arthrosis (Q74.3)

Autosomal recessive child type

Duchenne dystrophy abo Becker

Good for the scapula peroneal with early contractions [Emeri-Dreyfus]

Viklyucheni: incidence of myazovye dystrophy:

With refined morphological lesions of meat fiber (G71.2)

G71.1 Myotonic rods. Dystrophy is myotonic [Steiner]

Dominant success [Thomsen's]

[Becker's] recessive decline

Neuromyotonia [Isaacs]. Paramіotonіya vrodzhena. Pseudo-tone

If necessary, identifying a lykarskiy zazyb, wikkalo urazhennya, vikoristovuyut additional code of specific reasons (class XX).

Vrodzhena myazova dystrophy:

With specific morphological lesions of the meat

Disproportion of fiber types

Considerable [ailment of considerable size]

G71.3 Mitochondrial myopathy, not classified in other headings

G71.8 The first place to fight

G71.9 Unspecified blood pressure test Spadkova myopathy NOS

G72 Інші myopathies

Viklyucheni: congenital multiple arthrogriposis (Q74.3)

ischemic mucus infarction (M62.2)

G72.0 Lykarska myopathy

If necessary, identifying a likarskiy zakorystovuyut an additional code of specific reasons (class XX).

G72.1 Alcoholic myopathy

G72.2 Myopathy, viklicana with toxic speech

If necessary, identify a toxic word for a specific reason (class XX).

G72.3 Periodic parallel

Periodic parallich (family):

G72.4 Ignition myopathy, not classified in other headings

G72.8 Inspection of myopathies

G72.9 Myopathy, unspecified

G73 * Improvement of the nerve-muscle synapse and muscle in case of ailments, classify in the same headings

G73.0 * Myasthenic syndromes in endocrine ailments

Myasthenic syndromes with:

G73.2 * Inshi myasthenic syndromes with puffy lesions (C00-D48 +)

G73.3 * Myasthenic syndromes with other ailments, classification in the same headings

G73.5 * Myopathy in endocrine ailments

G73.6 * Myopathy in case of loss of speech exchange

G73.7 * Myopathy with other ailments, classified in the same headings

Cerebral palsy TA INSHI paralytic SYNDROME (G80-G83)

G80 Childish cerebral palsy

Includes: Little's ailment

Wicked: spadkovaya spastic paraplegia (G11.4)

G80.0 Spastic cerebral palsy Natural spastic paralysis (cerebral)

G80.1 spastic diplegia

G80.3 Diskinetic cerebral palsy. Athetoid cerebral palsy

G80.4 ataxic cerebral palsy

G80.8 The first type of infant cerebral palsy. Cerebral palsy syndrome

G80.9 Childish cerebral palsy Cerebral palsy NOS

G81 Hemiplegia

Note For the first coding of the ciu heading, go to and from only todi, if about hemiplegia (new)

(Discreet) to appear without additional clarification.

Viklyucheni: congenital that childish cerebral palsy (G80. -)

G81.1 spastic hemiplegia

G81.9 hemiplegia unspecified

G82 Paraplegia and tetraplegia

Viklyucheni: congenital or childish cerebral palsy (G80. -)

G82.1 spastic paraplegia

G82.2 Paraplegia, unspecified Parallels of both lower definitions of the NDU. Paraplegia (lower) NOS

G82.4 spastic tetraplegia

G82.5 Tetraplegia, unspecified Quadriplegia NOS

G83 Іnshі parallel syndromes

Note For the first coding of the heading, the heading is just like that, if you think about the overwhelmed state of affairs without further clarification. reasons for identifying cich stanіv, cyclic baths, be the reason.

Inclusions: paralich (povniy) (non-povniy), except for those specified in rubrics G80-G82

G83.0 diplegia of the upper cents. Diplegia (upper). Parallels of both upper cuts

G83.1 monoplegia of the lower picture. Parallich of the lower picture

G83.2 monoplegia of the upper pattern. Parallich of the upper film

G83.3 monoplegia, unspecified

G83.4 Chinese tail syndrome Neurogenic slash michur, dressings with Kinsky tail syndrome

Viklyuchenii: spinal slice michur NOS (G95.8)

G83.8 - Specification of paralytic syndromes Paralich Todd (gradual)

G83.9 Paralitic syndrome of unspecified

INSHI NERVOUS SYSTEM DISRUPTION (G90-G99)

G90 Rozladi of the vegetative [autonomous] nervous system

Excludes: autonomic nervous system disorder, alcohol wiclican (G31.2)

G90.0 Idiopathic peripheral autonomic neuropathy. Negligence, dressings with teasers of the carotid sinus

G90.1 Family design [Rail-Day]

G90.2 Horner's syndrome Bernard (-Gorner) syndrome

G90.3 Polysystemic degeneration Neurogenic orthostatic potency [Shay-Drager]

Included: Orthostatic NOS (I95.1)

G90.8 Інші pathways of the autonomic [autonomous] nervous system

G90.9 Rozlad of the autonomic nervous system, unspecified

G91 Hydrocephalus

Includes: dribbling of hydrocephalus

G91.0 Received hydrocephalus

G91.1 Obstructive hydrocephalus

G91.2 Hydrocephalus of normal vise

G91.3 Posttraumatic hydrocephalus, unspecified

G91.8 Other types of hydrocephalus

G91.9 Hydrocephalus, unspecified

G92 Toxic encephalopathy

If necessary, identify toxic speech

additional code of calling reasons (class XX).

G93 Brain damage

G93.0 Cerebral cyst. Arachnoid cyst. Parentcephalic city pribana

Viklyucheni: periventricular nabuta cyst of the new population (P91.1)

congenital cerebral cyst (Q04.6)

G93.1 Anoxic lesions of the brain, not classified in the bath in these headings

G93.2 Good internal cranial hypertension

Excluded: hypertensive encephalopathy (I67.4)

G93.3 Syndrome of stomatitis due to transferred viral ailments. Good mіalgicheskіy encephalomієlіt

G93.4 Encephalopathy, unspecified

G93.5 Cerebral stagnation

Obmezhennya> brain (stovbur)

Excludes: traumatic cerebral compression (S06.2)

Viklyucheni: bulging the brain:

G93.8 Іnshі refined lesions of the brain. Encephalopathy, due to the disorder

If necessary, identifying a specific factor will pick up the additional code of specific reasons (class XX).

G93.9 Brain lesion, unspecified

G94 * Інші lesion of the brain with ailments, classified in baths in the same headings

G94.2 * Hydrocephalus with other ailments, classified in the same headings

G94.8 * Іnshi clarification of lesions of the brain in diseases classified in the same headings

G95 Spinal ailments

G95.0 syringomyelia and syringobulbia

G95.1 Sudden mylopathy. Gostriy infarction of the spinal cord (embolic) (non-embolism). Spinal artery thrombosis. Hepatomielia. Nongenic spinal phlebitis and thrombophlebitis. Spinal cord abscess

Subacute necrotizing myelopathy

Vklyuchenii: spinal phlebitis and thrombophlebitis, but not pyogenic (G08)

G95.2 Spinal cord pressure, unspecified

G95.8 Іnshі specification of ailments of the spinal cord. "Spinal" section michur NOS

If necessary, identifying a specific factor will pick up the additional code of specific reasons (class XX).

Viklyucheni: neurogenic sečovy mіkhur:

nerve-muscle dysfunction of the cuticle without clues about the lesion of the spinal cord (N31. -)

G95.9 Spinal ailment, unspecified mylopathy NOS

G96 Central nervous system damage

G96.0 Termination of cerebrospinal birth [lekvorrhea]

Excludes: for spinal puncture (G97.0)

G96.1 Infections of the membrane of the brain, not classified in other headings

Meningeal growth (cerebral) (spinal)

G96.8 Inspection of the Central Nervous System

G96.9 Exercise of the central nervous system, unspecified

G97 Damage to the nervous system due to medical procedures, which cannot be classified in other headings

G97.0 End of cerebrospinal line with spinal puncture

G97.1 Insha reaction to lumbar puncture

G97.2 Intracranial hypertension for bypassing

G97.8 - Damage to the nervous system due to medical procedures

G97.9 Rozlad of the nervous system for medical procedures, unspecified

G98 - Damage of the nervous system, not classified in the other headings

Breakdown of the nervous system and the NOS

G99 * Інші damage of the nervous system with ailments, classify baths in the same headings

G99.0 * Vegetative neuropathy in endocrine and metabolic ailments

Amiloid autonomic neuropathy (E85. - +)

Diabetic autonomic neuropathy (E10-E14 + from the fourth sign 4)

G99.1 * Infringement of autonomic [autonomic] nervous systems for some ailments classified in others

G99.2 * Mylopathy with ailments, classified in the same headings

Syndrome of suppression of anterior spinal and spinal artery (M47.0 *)

G99.8 * Inspection of damage to the nervous system in diseases classified in the other headings

What is the value of the treatment of polyneuropathy according to ICD-10?

The polyneuropathy code according to ICD-10 will be different for fallowness, depending on the type of disease. Given the uncomfortable mind of a pathological camp, with any hostilities, a nerve in the body of a man. It also manifests itself in the form of parallels, paresis, problems with trophic tissue and vegetative type growths.

Axonal polyneuropathy of the lower symptoms or of the lower parts of the body develops when the communication processes are broken. For example, an affliction can be manifested when mish, mercury, lead and other words get into the body. In addition, the alcoholic form can be included until the end. For the interruption of polyneuropathy, there is a hostility, pidgostroy, chronic, recurrent.

See the onset of the axonal polyneuropathy:

1. 1. Gostra form. To develop with a stretch of decilkoh dib. The nerve cut is tied with a strong intoxicating organism through the infusion of methyl alcohol, mish'yak, mercury, lead, foul gas and other spoluk. This form of pathology can be no more than 10 dB. Therapy is carried out under the control of a doctor.
2. 2. Subacute. Vaughn grows out with a stretch of decіlkokh tizhnіv. It is typical for toxic and metabolic species. Oduzhati viyde only through the clump of miscellaneous.
3. 3. Chronic. It grows in a stretch of a trivial advance for an hour, for more than 6 months. Such a type of pathology is progressing, as there is a lack of vitamin B12 or B1 in the body, and also lymphoid, cancer, pukhlina, and diabetic diabetes develop.
4. 4. Recurrent. There can be a lot of sick people more than once, and it can be manifested by a stretch of rockets, albeit periodically, and not permanently. It is often possible to achieve this in case of alcoholic forms of polyneuropathy. Qia ailment vvazhaє to be even more insecure. Growing up, only when Lyudin lived on a lot of alcoholic beverages. At the same time, it is not only the amount of alcohol that plays a great role, but also its quality. Tse is disgustingly pouring into the zalny camp of the healthy people. During the period of therapy, it is categorically to abstain from living alcohol. Due to the alcoholic content, it may be necessary to use it as a matter of course.

The demiєlіnіzuyucha form is characteristic of the Bari-Gin syndrome. Tse pathology of the ignition type. Її provoke ailments, infections. With a lot of people, there is an operative type of pain in the legs and weakness in the muscles. The price of the characteristic features of the ailment. After the weak health, symptoms of the sensory form of the ailment appear in an hour. The development of the whole ailment can be carried out in months.

If the patient has diphtheria-type polyneuropathy, then in a couple of days there will be enemies of the cranial nerves. Through the tse of awesome mova, people are important to grow, kovtati їzhu. The integrity of the phrenic nerve will also deteriorate, so it is important for people to dichati. Parallich of kintsivok insisted only through the month, and the whole hour the sensitiveness of the hands would be ruined step by step.

Also, there is a classification of polyneuropathy for provocative officials:

1. 1. Toxic. The qia form is manifested through the detachment of the organism by the chimneys of the chimneys. It may not be just a mish'yak, mercury, lead, ale and a pobutova chemistry. In addition, the toxic form manifests itself with trivial alcoholic deposits in chronic forms, as it is badly infused into the camp of the nervous system and cause malnutrition in the robotic organs of children. Another type of toxic polyneuropathy is diphtheria. Vaughn manifest itself as an accelerated pislya diphtheria. Zazvychay to grow up to do the trick at the grown-ups. The pathology is characterized by small changes, which are linked to the functions of the nervous system. For example, it’s easy to lose weight with the cleanliness of fabrics, and it’s awesome for rukhov’s function. Lіkuvati similar polyneuropathy is guilty of only lіkar.
2. 2. Ignite. Tsey razvidovy zvoryuvannya develop only for the development of ignition processes in the nervous system. At the same time, there is an inappropriate vision, it is in the legs and hands. Maybe the building is ruined and talked about. If any of these symptoms appear, it is necessary to immediately return to the hospital.
3. 3. Allergic. The qi form is developed on aphids of toxicity with methyl alcohol, mish'yak, foul gas and phosphorus-organic fluids. A great role is played by the chronic form of intoxication with the help of others. Unfriendly є prognosis for diabetics, diphtheria and avitaminoses. To achieve the often allergic form of getting sick grows through the trivial implantation of any medication.
4. 4. Traumatic. Tsey rizvid z'yavlyatsya through the removal of serious injuries. Symptoms will only be shown by the onset of the onset of deciduous movements. Name the head symptom є deterioration of the rukhovaya function. Even more important before the hour of exercise є exercise and exercise therapy.

Vidіlyayut and inshi, less widening of the form of polyneuropathy.

For an ailment such diabetic polyneuropathy: code for MKH-10, clinical picture and methods of treatment

Polyneuropathy is a complex of ailments, which includes the so-called multiple lesions of peripheral nerves.

The disease is encouraged to move into the so-called chronic form and the long way wider, so that the process of combining the enemy itself with other fibers and, by and large, overflows into more large spikes.

This pathological camp, called diabetic polyneuropathy ICD-10, encrypts and grows in fallowness as a result of walking, overcoming ailments on the onset of the group: ignition and polyneuropathies. So also diabetic polyneuropathy according to ICD?

So tse take?

Polyneuropathy is the so-called acceleration of the diabetic diabetes, the whole essence of the polyagus is in the generalized accelerated nervous system.

Nerve reduction in polyneuropathy

Call yourself out to show yourself through a significant interval of an hour, which has passed at the moment of diagnosis of damage in the robotic endocrine system. More precisely, the disease can appear in twenty five rockets in the form of development of problems with the insulin in people.

Ale, bully vipadki, if a sickle of a bullet has been detected in patients of endocrinologists already through five years since the moment of revealing pathological diseases of the lump. Rizik zvoriti - the same for children with diabetes, as the first type, so and the other.

Causes of the blame

As a rule, in case of a trivial interruption of the disease and the frequent consumption of the tsukru, the breakdown of the metabolic processes in all organs and systems of the body is diagnosed.

Moreover, the first suffering is the nervous system itself. As a rule, nerve fibers to liven up blood-bearing judges.

We are trivial in pouring in carbohydrates because it is called the destruction of nerves. As a result of which stench falls into the camp of hypoxia, as a legacy, the primary symptoms of illness appear.

With a slight interruption and frequent decompensation, it is significant to accelerate the apparent problems with the nervous system, which step by step inflate a nonvolatile chronic character.

Diabetic polyneuropathy of lower symptoms according to ICD-10

The same diagnosis most often smells the children who are suffering from diabetes.

Denmark of ailment is hostile to the organism of todi, if the peripheral system and fibers are destroyed. There can be provocations by the very different factors.

As a rule, people of the middle age were strong before, in the first place. Most of the time, some people get sick more often. It is also obvious that polyneuropathy is not common in children of preschool and adulthood.

Diabetic polyneuropathy, code for MKH-10, which is E10-E14, invoke enemies of the very top and bottom of people. As a result, responsiveness, praiseworthiness decreases, the quality becomes asymmetric, and blood circulation also decreases. It seems to me that the head particularity of the whole ailment is that those who are guilty are widening in every way, hostile to the very same nerve fibers. It is not marvelous for those who are calling, for those who suffer from their feet.

signs

Diabet is afraid of the whole cat, yak will burn!

It is necessary for all to have it.

Sickness, manifested overwhelmingly on the lower symptoms, volodya great number of symptoms:

• feeling a strong oniminnya in the legs;
• swelling of the feet and nig;
• unbearable pain and prickly vision;
• weakness in meat;
• increase or decrease in sensitivity of pictures.

The cutaneous form of neuropathy leads to the following symptoms:

1. diabetic at the first stages. Vaughn is characterized by some of the lower cues, a lot of pokryuvannya and strong writing in them. The presence of ice is smudged in the feet, gomilkovy soblobnyh slopes, as well as in litkovye ointments. As a rule, at night the symptoms become more severe and swelling;
2. diabetic at lower stages. With the appearance of such troubling symptoms: unbearable beating in the lower symptoms, as it can also appear in the country of calm, weakness, atrophy of the muscles and the symptoms of the pigmentation of the school. We will proceed with the development of the sickness, the stink will burn, as a result of which the stench will become more delicate, or will atrophy. Also, in a sick person, a diabetic foot is called so called: it means to grow in size, there is flatness, ankle deformity and neuropathic bulges develop;
3. diabetic encephalopolyneuropathy. For some of the following signs are characteristic: how not to go through severe headaches, instantaneous and increased stomachache;
4. toxic and alcoholic. Їy characteristic symptoms such as severe swelling of the symptoms: sudomy, oniminnya nig, not impaired sensitivity of the feet, weakening of the tendon and reflexes of the muscles, change to the bluishness of the scalp, I will curl on the bluishness of the hair of the hair, change As a heritage, the trophies take off and the swelling of the nig.

diagnostics

Oskilka one type of dosage cannot show again the picture, then diagnostics of diabetic polyneuropathy is carried out using the ICD-10 code for additional decals of popular methods:

As a rule, the first method to follow-up is to take a detailed look at a number of people: a neurologist, surgeon and endocrinologist.

The first doctor to deal with the development of specific symptoms, such as: blood pressure in the lower symptoms and sensitivity, the presence of all the necessary reflexes, conversion to the increase in the size of the machine.

As far as laboratory dosages are concerned, they include: analysis of blood plasma, glucose concentration in blood plasma, cholesterol, as well as the level of toxic speech, which can be found in the body when it is suspected of having toxic neuropathy.

And the axis is instrumental diagnostics of the presence of diabetic polyneuropathy in the organism of a patient according to ICD-10 May on the basis of MRI, as well as electroneuromyography and biopsies of nerves.

lykuvannya

It is important to memorize, but lіkuvannya is guilty of being complex іmіshanim. Vono, it is not uncommon to include in itself perennial drugs, which are direct on the entire process of development.

Even more importantly, the treatment included the intake of these drugs:

1. vitamini. Smells of guilt come into the body at once with food products. The workers use the transport of impulses along the nerves, and also block the negative effects of glucose on the nerves;
2. alpha-lipoic acid. Won the purchased zukru in the nerve tissue, activating the singing groups of enzymes in the cells and changing the nerve damage;
3. antibiotic preparations;
4. іngіbіtory aldose reductase. The stench to stand at the plant is one of the number of transformations of zukru in the blood, which change by itself and infuse on the nerves of the end;
5. actovegin. Winning glucose vikorizannya, will dye microcirculation of blood in arteries, veins and capillaries, which nudge nerves, as well as transferring nerve cells;
6. Calcium and Calcium. At the same time, the power of change of judgment and oniminnya in the pictures of the people may cause the power of change;
7. antibiotics. You can only get it right, if there is a risk of gangrene development.

In addition, as the very form of diabetic polyneuropathy ICD-10 is revealed, the eligible doctor recognizes a professional treatment, as I increase the symptoms of the disease. At the same time, it is possible to help on the outside of the vil_kuvannya.

Even more importantly, it is important to reduce the amount of blood in the blood, and only then to start testing for diabetic polyneuropathy according to the ICD. If you do not kill, then all efforts will be absolutely ineffective.

It is even more important in the case of toxic forms of increasing alcoholic drinks and after being able to take advantage of the suvorian children. A liquid drug is obviously guilty of special medications that cause microcirculation of blood and overload blood clots. Even more importantly, get out of the mess.

Video by topic

Candidate of Medical Sciences about polyneuropathies in diabetics:

You can be intelligent with the help of information, you can see it in the statistic, diabetic neuropathy can be done well. Naygolovnishe - not starting the process. The ailment can be very swirling of symptoms, as it is important not to mention it, so with a reasonable approach to it, you can end up quickly getting confused. For the first symptoms of anxiety, it is important to go through medical procedures, to confirm the transfer of diagnosis. As soon as it goes, you can go to the treatment of ailment.

• Stabilizu rivn tsukru nadovgo
• Introduce the revolving to the insulin in the slurry plant