Mkb 10 codi hvorob anemia. Fatal deficiency anemia

anemia- the price of inconsistency of the proportion of hemoglobin in the blood of a person to the criteria adopted by the All-Holy Organization of Health Protection for a particular type and status. The term "anemia" is not a diagnosis of anemia, but only because of abnormal changes in the blood analysis.

The code for the international classification of ailments MKH-10: zalizodeficitna anemia - D50.

Most widening anemia as a result of blood loss and hypertension anemia:

1. Anemia as a result of blood loss may be a viclican with trivial menstruation, bleeding in herbal tract and every day, traumas, operations, oncological illnesses.
2. Fatal deficiency anemia to assert in the result of a deficiency in virobnism by the organism of red blood cells

Reasons and factors

Among the factors that increase the risk of getting anemic, you see:

• lack of implantation of hair, vitamins and minerals;
• pogany kharchuvannya;
• waste blood through trauma or surgery;
• ailments nirok;
• diabet;
• rheumatoid arthritis;
• VIL / SNID;
• igniting intestinal ailments (including Crohn's ailment);
• getting sick of pechinki;
• heart failure;
• ailing thyroid gland;
• anemia of sickness, vicious infection.

Khibna dumka, how anemia is found only when there is ailment.

There are more reasons for this:

Stages and types of anemia

1. lungs- the amount of hemoglobin in the warehouse is 90 g / l and food;
2. middle severity grade - hemoglobin 70-90 g / l;
3. important anemia - hemoglobin lower than 70 g / l with the norm for women 120-140 g / l, for cholovikiv - 130-160 g / l.

• Anemia through deficiency of hair... Women need an hour of vaginosity, menstruation and lactation during the period of lactation. Tomato deficiency anemia is often consumed during the whole period.
  Likewise, the child itself and the child vimagak bagato zaliza. This kind of anemia can be found in the form of alcohol tablets or syrups.
• megaloblastic anemia winery as a result of hormone deficiency in the thyroid gland, liver ailments and tuberculosis. The whole type of anemia, viklicanii not too much vitamin B12 and folate acid. early diagnosis and the disease is even more important for the ailing megaloblastic anemia.
  Weakness, in volume, Onimіnnya hands, bіl і sorrowful move, zadishka - broadening of the scarg type of ailment.
• Chronic infection anemia winery through the failure of the cystic marrow, with tuberculosis, leukemia, and as a result of the intake of such cases, in warehouses and toxic speech.
• Seredzemomorska anemia(Ailments, also in the form of thalassemia) Visoka sickness of the cymbal type is promoted to occur in the Italians and Greeks. On the cob stage, the symptoms are the same as in case of anemia through a deficiency of hair.
  In the world of progressing ailments Sposter zhovtyanitsya, to achieve anemia as a result of ailments nirok and growth of the spleen. Thalassemia is susceptible to blood transfusions.
• sickle cell anemia the chain of ailment spadiosis when the structure of hemoglobin in the blood appears from normal indicators... The erythrocyte takes the form of a pivmisyatsya, the hour of your life is even short. This type is promoted to representatives of the Black Race. Carrier of the gene for anemia in women.
• aplastic anemia the tse of the destruction of the worm blood cells in the cystic marrow. The reason may be the state of the vaporization of luscious words, such as benzene, mish'yak, injecting radioactivity. The blood level of platelet cells also decreases.
  Aplastic anemia є polycythemia The number of erythrocytes in the blood is growing by 2 times. The shkira of the patient grows old and can be helped to advance the arterial grip. The reason for this is marriage. Likuєt the tsya ailment by the way of blood for the body of the people.

Who can get anemia?

Anemia ailing, as hostile to all vіkovі and ethnіchnі groups, races.

• Deyakі kids in the first rіk life Risk of anemia is increased through deficiency of hair loss. There are many people and children who have been breastfeeding for years with a little hair. In such people, anemia develops in the first 6 months.
• Children from one to two years old until the development of anemia... Especially, if the stench smells rich in cow's milk and does not eat a lot of hair. At the warehouse of cow's milk, there is a lot of money for the growth of a child. milk replacement baby up to 3 rocky next year grab a lot of food. Cow's milk can also be absorbed into the body.
• Prelіdniki prodovzhuyut vivchati yak anemia pouring in on grown-ups. More than ten hundred years old are constantly found in the country of easy anemia. Most of the people in the world have little medical diagnosis.

Signs and symptoms

The most common symptom of anemia in the volume. People see for themselves weary and win.

Other signs and symptoms of anemia include:

• difficulty in dikhannya;
• zamorochennya;
• headaches;
• cold feet and hollows;
• bile at the breasts.

Symptoms can appear through those that have become more important to the heart to pump the bagatu on a kisen shelter in til.

In a mild and middle stage of anemia (zalizodeficitnoy type) symptoms є:

• bazhannya z'isty foreign subject: earth, ice, vapnyak, starch;
• trischini in kuchiny company;
• razdratovany mov.

Signs of folate deficiency:

• carry;
• depression;
• swollen і red tongue;

Symptoms of anemia due to vitamin B12 failure:

• pokryuvannya and loss of sensitivity in the upper and lower concepts;
• folding in pink and blue color;
• embryo і bіl in the larynx;
• waste at vase;
• pochorninnya shkiri;
• carry;
• depression;
• reduction of intellectual functions.

accelerating

A lykar in case of a naked diagnosis is guilty of overcoming anemia:

1. Patients may have arrhythmias- the problem with the speed of the heart and the rhythm of the heart is very fast. Arrhythmia can lead to heart failure and heart failure.
2. anemia can You can also bring some organs to life until they are finished: shelter cannot be provided with a sufficient amount of sourness.
3. at cancer patients і VIL / SNID ailment can weaken the body, і change the result as a result of the disease.
4. adjustments of risik the diagnosis of anemia with nirok ailments, in patients with heart problems.
5. Deyaki tipi anemia to be diagnosed with a lack of co-existence of a child or an overwhelming loss of water in the body. Stronger anger is the cause of blood loss.

diagnostics

Likar is guilty of knowing the history of the ailment, but because of the low type of ailment. Wien can feed the patient about the signs of anemia, which are perebuva wines on the day.

Physical regulation to become:

1. listening to the rhythm of the heart and regularity of the sound;
2. vimir to the size of the spleen;
3. the presence of pelvic abo-rectal bleeding.
4. laboratory analyzes can help determine the type of anemia:
   • zagalny blood analysis;
   • hemograms.

Hemogram test for hemoglobin and hematocrit in blood. Low hemoglobin і hematocrit є familiar anemia. Normal values ​​vary in the fallowness of races and populations.

Інші tests and procedures:

• electrophoresis hemoglobin the number of different types of hemoglobin in the blood.
• Vimiruvannya of reticulocytes- a chain of young red blood-blooded children in the blood. The whole test will show the swiftness of the cystic brain tissue formation.
• Testi vimіryuvannya zaliza in the blood- the cost of living in the place of the meeting, transmission, and the sound of blood.
• Iakshcho lykar undergo anemia through blood loss, You can get an analysis for the appearance of a bleeding disorder. Propose an analysis of feces for blood count in feces.
  Yakshcho shelter є, endoscopy is required: rigidity internal side herbal system and a small camera.
• can you get to know also the analysis of the cystic brain.

How can you catch anemia?

Likuvannya anemia lie in the cause, importance and type of ailment. By the method of lіkuvannya є increasing the sourness in the blood, the multiplication of chervony clitin and the growth of hemoglobin.

Hemoglobin tse block, which is the transport of pink to the floor for additional travel.

Changes and additions to the harvesting regime

Zalizo

Organism needs to formulate hemoglobin. Tilo glaze zalizo from meat is lighter, less from vegetables and other products. To get rid of anemia, you need more meat, especially red meat (yalovychina or pechinka), as well as chicken, іndichka and seafood.

Крім м'яса, zalizo є in:

vitamin B12

Low level of vitamin B12 can lead to malignant anemia.

Dzherelami vitaminu B12 є:

• grain;
• chervone meat, liver, poultry, riba;
• eggs and dairy products (milk, yogurt and sir);
• sovі nої on the basis of zalіza і vegetarian products, with vitamins B12.

Folic acid

The body needs folate acid, to produce new cells and cleanse them. Folic acid is necessary for vaginal women. Wonderful because of anemia and additional help to healthy development of the fetus.

Good dzherelo food products in folic acid є:

• Khlib, macaroni virobi, rice;
• spinach, dark green leafy vegetables;
• dry bobies;
• oven;
• egg;
• bananas, oranges, orange juice and fruits and juices.

Vitamin S

Win to help the body go to the zalizo. Fruits and vegetables, especially citrus fruits, are a good jerel to Vitamin C. Fresh and frozen fruits and vegetables are higher than Vitamin C, less in canned products.

Vitamin C bagati kivi, polnitsya, din, broccoli, pepper, Brussels sprouts, Tomato, potato, spinach, radish.

Medication

Lykar can be indicative of the main causes of anemia and reduction of a number of red blood cells in the body.

Tse mozhut buti:

• antibiotics for the treatment of infections;
• hormones for overwhelming menstrual bleeding in young girls and women;
• piece erythropoietin for stimulation of erythrocyte viroblasting.

operations

As the anemia has grown into an important stage, an operation may be necessary: ​​transplantation of stem-boron blood cells and blood cells, blood transfusion.

The transplantation of stovbrovykh cells is available to replace the girls with an ailing healthy donor. Stovburovі cells are located in the cystic marrow. The transmission of cells is through a tube inserted into a vein in the breasts. The process is similar to blood transfusion.

surgical engagement

at clutter up the life bleeding in the body, wick anemia, Necessary surgical involvement.

For example, anemia at swirling ailments shlunka abo to cancer of the intestine vimagayut surgical engagement to prevent bleeding.

prophylaxis

Deyakі type of anemia can be zapobіgti, vivayuyu їzhu, bagatu zalizom and vitamins. korynno accept food additives pid hour of the day.

Important! For women who want to get thinner and smaller in size with additional supplements from zalizom and Vitamin complexesє binding!

For the main treatment of anemia, it is necessary to adjust the connections with your eligible doctor and regularly change the blood warehouse.

Iakshko ailment having calmed down the evil type of anemia, the abuse and prevention is guilty of the trivia rocks. We cook until you need it.

Anemia in children and young people

Chronic illness, deficiency of health and bad food can lead to anemia. Illness is often supervised by other health problems. Thus, the signs of the symptoms of anemia are often not obvious.

Obov'yazkovo varto turn up to the drug, as the symptoms of anemia are spared, or the person will be overused for the day. Blood transfusion or hormonal therapy can be done. As soon as anemia is diagnosed, it is possible to develop vilikuvati.

Class III. Ailments of blood, hematopoietic organs, and the surrounding of damage to the results of the immune mechanism (D50-D89)

Victims: autoimmune ailments (systemic) NOS (M35.9), near the village, occurring in the perinatal period (P00-P96), accelerated vaginosity, lower and lower periods of the disease (O00-O99), deformity - Q99), endocrine ailments, problems with eating and failing to exchange diseases (E00-E90), sickness, sickness, human immunodeficiency virus virus [VIL] (B20-B24), traumas, failure calling reasons(S00-T98), new findings (C00-D48), symptoms, signs and symptoms of deterioration according to norms, revealed in clinical and laboratory diagnoses, not classified in other headings (R00-R99)

Tsey class to take revenge on the offensive blocks:
D50-D53 Anemias, knitted from food
D55-D59 Hemolytic anemias
D60-D64 Aplastic and insignificant anemias
D65-D69 Loss of laryngeal blood, purpura and other hemorrhagic beds
D70-D77 Other ailments of the blood and hematopoietic organs
D80-D89 Damage caused by the immune mechanism

The third category is as follows:
D77 Infringement of blood and hematopoietic organs with ailments classified in the same headings

ANEMI, CONNECTED TO LIVES (D50-D53)

D50 Zalizodeficitna anemia

Included: anemia:
... cyderopenic
... hypochromic
D50.0 Zalizodeficitna anemia is secondary in cases of blood loss (chronic). Post-hemorrhagic (chronic) anemia.
Wikipedia: posthemorrhagic anemia (D62) congenital anemia during fetal hemorrhage (P61.3)
D50.1 Syderopenic dysphagia. Kelli-Paterson syndrome. Plummer-Winson syndrome
D50.8Іnshі zalіzodeficitnі anemia
D50.9 Zalizodeficitna anemia, unspecified

D51 Vitamin-B12 deficiency anemia

Victims: Vitamin B12 deficiency (E53.8)

D51.0 Vitamin-B12-deficiency anemia due to deficiency internal official.
anemia:
... Addison
... Birmer
... pernicious (vrogena)
There is a lack of an internal official
D51.1 Vitamin-B12 deficiency anemia due to vibrational damage due to wetting Vitamin B12 with protein.
Imerslund (-Gresbeck) syndrome. Megaloblastic spadkov anemia
D51.2 Deficiency of transcobalamin II
D51.3Іnshi vitamins-B12-deficient anemias, tied to food. anemia of vegetarians
D51.8Іnshi vitamins-B12-deficiency anemias
D51.9 Vitamin-B12 deficiency anemia, unspecified

D52 Folio-water deficiency anemia

D52.0 Folio-water deficiency anemia, tied to food. Megaloblastic alimentary anemia
D52.1 Foliodeficiency anemia is medication. If necessary, identify the license.
vikoristovuyt additional code of calling reasons (class XX)
D52.8 Follicle water deficiency anemia
D52.9 Folio-water deficiency anemia is unspecified. Anemia caused by the lack of proper functioning in the organism of folic acid, NOS

D53 Inshi anemia, knitted with a crochet

Included: megaloblastic anemia,
nom B12 abo folate

D53.0 Anemia due to lack of bile. Anemia due to lack of amino acids.
Orotational anemia
Viklyuchenii: Lesch-Nihen syndrome (E79.1)
D53.1Інші megaloblastic anemia, not classified in these headings. Megaloblastic anemia NOS.
Excluded: ailment Di Gul'lmo (C94.0)
D53.2 Anemia, surrounded by scurvy.
Vicious: scurvy (E54)
D53.8Інші clarification of anemia, tied with food.
Anemia, associated with a deficiency:
... міді
... molybdenum
... zinc
Viklyuchenya: lack of food without a puzzle about
anemia, such as:
... midi deficiency (E61.0)
... molybdenum deficiency (E61.5)
... zinc deficiency (E60)
D53.9 Anemia, tied to food, unspecified. Simple chronic anemia.
Excluded: anemia NOS (D64.9)

Hemolytic anemia (D55-D59)

D55 Anemia due to enzyme damage

Excluded: enzyme deficiency anemia, vicylican lyceum diseases (D59.2)

D55.0 Anemia due to lack of glucose-6-phosphate dehydrogenase [G-6-PD]. Favism. G-6-PD-deficiency anemia
D55.1 Anemia caused by those who suffer from glutathione metabolism.
Anemia due to lack of enzymes (with the guilt of G-6-PD), tied from hexose monophosphate [HMP]
shunt metabolic path. Hemolytic non-spherocytic anemia (spadkov) type 1
D55.2 Anemia caused by damage to glycolytic enzymes.
anemia:
... hemolytic non-spherocytic (spadkov) type II
... due to lack of hexokinase
... due to lack of pyruvate
... due to lack of triose phosphate isomerase
D55.3 Anemia due to impairment of nucleotide metabolism
D55.8Інші anemias in cases of enzyme powder
D55.9 Anemia due to enzyme damage, unspecified

D56 Thalassemia

D56.0 Alpha talasemia.
Fetal dropsy, surrounded by hemolytic ailments (P56. -)
D56.1 Beta talasemia. Anemia Kuli. Beta-talasemia is important. Sickle-cell beta-thalassemia.
thalassemia:
... promіzhna
... great
D56.2 Delta-beta-thalassemia
D56.3 Nosystvo signs of talasemiya
D56.4 Spadkovo persistence of fetal hemoglobin [NPFG]
D56.8іnshі thalassemias
D56.9 Thalassemia, unspecified. Seredzemomorska anemia (s)
Thalassemia (mala) (zmіshana) (from the least hemoglobinopathy)

D57 sickle-shaped disease

Viklyucheni: іnshі hemoglobinopathies (D58. -)
sickle-clitinna beta-thalassemia (D56.1)

D57.0 Sickle-cell anemia with crisis. Hb-SS ailment with crisis
D57.1 Sickle-cell anemia without crisis.
Crescent-shaped (e):
... anemia)
... ailment) NOS
... ruin)
D57.2 Subordinate heterozygous sickle-cell disease
ailment:
... Hb-SC
... Hb-SD
... Hb-SE
D57.3 The nosystvo is a sign of sickle-clitine. Nose to hemoglobin S. Heterozygous hemoglobin S
D57.8Інші crescent-shaped disease

D58 Lowest hemolytic anemia

D58.0 Spasmodic spherocytosis. Akholurichna (simeina) zhovtyanytsya.
Vrodzhena (spherocytic) hemolytic zhovtyanitsa. Mankivsky-Shoffard syndrome
D58.1 Spadkovy elliptocytosis. Ellitotsitosis (congenital). Ovalocytosis (congenital) (decline)
D58.2Інші hemoglobinopathies. Abnormal hemoglobin NOS. Anemia with Heinz's children is born.
ailment:
... Hb-C
... Hb-D
... Hb-E
Hemolytic ailment, zoomed with unstable hemoglobin. Hemoglobinopathy NOS.
Viklyucheni: familyna polycythemia (D75.0)
ailment Hb-M (D74.0)
decline in persistence of fetal hemoglobin (D56.4)
polycythemia, tied with visota (D75.1)
methemoglobinemia (D74. -)
D58.8Інші clarification of the decrease in hemolytic anemia. stomatocytosis
D58.9 Spadkova hemolytic anemia, unspecified

D59 Nabuta hemolytic anemia

D59.0 Medication autoimmune hemolytic anemia.
If necessary, identify the drug as a vicorist for the additional code of reasons (class XX).
D59.1Інші autoimmune hemolytic anemia. Autoimmune hemolytic ailment (cold type) (heat type). Chronic ailments
"Holodova agglutinovaya":
... ailment
... hemoglobinuria
Hemolytic anemia:
... cold type (secondary) (symptomatic)
... heat type (secondary) (symptomatic)
Victims: Evans syndrome (D69.3)
hemolytic ailment of fetus and neonatal (P55. -)
paroxysmal cold hemoglobinuria (D59.6)
D59.2 Medication non-autoimmune hemolytic anemia. Enzyme-deficient anemia medication.
If necessary, identifying a likarskiy zakorystovuyut an additional code of specific reasons (class XX).
D59.3 Hemolytic-uremic syndrome
D59.4Інші non-autoimmune hemolytic anemia.
Hemolytic anemia:
... mechanical
... microangiopathic
... toxic
If necessary, identify the cause of the viciousness of the additional code of the underlying reasons (class XX).
D59.5 Paroxysmal nichna hemoglobinuria [Markiafavi-Mikele].
D59.6 Hemoglobinuria due to hemolysis caused by other underlying causes.
hemoglobinuria:
... vid navantazhennya
... Marshova
... paroxysmal kholodova
Excludes: hemoglobinuria NOS (R82.3)
D59.8Іnshi nabuty hemolytic anemias
D59.9 Pridbana hemolytic anemia is unspecified. Idiopathic hemolytic anemia is chronic

Aplastic TA INSHI ANEMIS (D60-D64)

D60 Nabuta pure red cell aplasia (erithroblastopenia)

Included: red cell aplasia (dribbana) (grown-up) (with Tim)

D60.0 Chronic dribbling pure red cell aplasia
D60.1 Minuscha is dribbled pure red cell aplasia
D60.8Іnshі nabutі pure red cell aplasia
D60.9 Pridbana is pure red cell aplasia, unspecified

D61 Aplastic anemias

Exclusion: agranulocytosis (D70)

D61.0 Constitutional aplastic anemia.
Aplasia (pure) red cell:
... vrogena
... child
... pervinna
Blackfen-Daymond syndrome. Semeina is hypoplastic anemia. Anemia Fanconi. Pancytopenia with vads development
D61.1 Medication is aplastic anemia. If necessary, identify the license.
vikoristovuyt additional code of calling reasons (class XX).
D61.2 Aplastic anemia;
If it is necessary to identify the cause of the vikoristovuyut additional code of the underlying reasons (class XX).
D61.3 Idiopathic aplastic anemia
D61.8Інші clarification of aplastic anemia
D61.9 Aplastic anemia is unspecified. Hypoplastic anemia NOS. Hypoplasia of the cystic marrow. panmieloftiz

D62 Gostra post-hemorrhagic anemia

Excluded: congenital anemia due to fetal haemorrhage (P61.3)

D63 Anemia with chronic ailments, classified in the same headings

D63.0 Anemia with new conditions (C00-D48 +)
D63.8 Anemia with others chronic ailments, Classified in the same headings

D64 Інші anemia

Viklyucheni: refractory anemia:
... NOS (D46.4)
... too much blast (D46.2)
... transformation (D46.3)
... with sideroblasts (D46.1)
... without sideroblasts (D46.0)

D64.0 Spadkova sideroblastic anemia. Hypochromic sideroblastic anemia is associated with the disease
D64.1 Secondary sideroblastic anemia in conjunction with other illnesses.
If necessary, identifying a pre-existing code.
D64.2 Secondary sideroblastic anemia, viclikana with drugs and toxins.
If it is necessary to identify the cause of the vikoristovuyut additional code of the underlying reasons (class XX).
D64.3Іnshі sideroblastic anemia.
Sideroblastic anemia:
... NOS
... peridoxin-responsive, not classified in these headings
D64.4 Vrodzhena diseritropoetic anemia. Dyshemopoietic anemia (born).
Vicious: Blackfen-Daymond syndrome (D61.0)
ailment Di Gul'lmo (C94.0)
D64.8Інші clarification of anemia. Childish pseudo leukemia. Leukoeritroblastic anemia
D64.9 anemia, unspecified

Destruction of the throat of blood, purpura TA INSHI

Hemorrhagic STANE (D65-D69)

D65 Dissemination of the internal vascular laryngeal [defibrillation syndrome]

Afibrinogenemia pridebana. coagulopathy
Diffuse or disseminated internally
Fibrinolytic bleeding of the dribble
purpura:
... fibrinolytic
... bliscavic
Vklyucheniy: syndrome defibrіnatsіі (scho accelerated):
... in newborn (P60)

D66 Substitious factor VIII deficiency

Factor VIII deficiency (with functional impairment)
hemophilia:
... NOS
... A
... classic
Inclusion: Factor VIII deficiency in vascular lesions (D68.0)

D67 Subsidious factor IX deficiency

Khvoroba Kristmas
deficiency:
... factor IX (with functional impairment)
... thromboplastic component of plasmas
hemophilia B

D68 Disease of the throat

Viklyucheni: speed up:
... abortion, post-mortem or molar vaginism (O00-O07, O08.1)
... vagіnіst, curtains and inspirational period (O45.0, O46.0, O67.0, O72.3)

D68.0 Villebrand's ailment. Angiohemophilia. Deficiency of factor VIII in cadaverous lesions. Sudden hemophilia.
Victory: Spadkov capillary lameness (D69.8)
factor VIII deficiency:
... NOS (D66)
... functional impairment (D66)
D68.1 Recessive deficiency of factor XI. Hemophilia C. Deficiency of thromboplastin transplanted plasma
D68.2 Spasmodic deficiency of the other factors of the throat. Vrodzhena afіbrinogenemia.
deficiency:
... AC-globulin
... proaccelerina
Factor deficiency:
... I [fibrinogen]
... II [prothrombin]
... V [labial]
... VII [stable]
... X [Stuart-Prower]
... XII [Hageman's]
... XIII [fibrіnstabіlіzing]
Disfibrinogenemia (vrodzhena). Ovren's ailment
D68.3 Hemorrhagic damage caused by anticoagulants circulating in the blood. Hyperheparinemia.
Advice to the zmist:
... antithrombin
... anti-VIIIa
... anti-IXa
... anti-Xa
... anti-XIa
If necessary, identification of vikoris anticoagulant vikoristovyt additional code of reasons
(Class XX).
D68.4 Nabutius deficiency of the factor of hypoglottism.
Laryngeal factor deficiency for some reason:
... disease of pechinki
... lack of vitamins K
Excluded: lack of vitamin K in a newborn (P53)
D68.8Іnshi clarification of the deterioration of the throat. The presence of the ingibitor of the systemic heart
D68.9 Unspecified zgortania

D69 Purpura and other hemorrhagic beds

Viklyucheni: good hypergammaglobulinemic purpura (D89.0)
cryoglobulinemic purpura (D89.1)
idiopathic (hemorrhagic) thrombocythemia (D47.3)
bliscavic purpura (D65)
thrombotic thrombocytopenic purpura (M31.1)

D69.0 Allergic purple.
purpura:
... anaphylactoid
... Henoch (-Shenlein)
... non-thrombocytopenic:
... hemorrhagic
... idiopathic
... sudinna
allergic vasculitis
D69.1 Yakisnі thrombocyte defects. Bernard-Sul'e syndrome [hypertrophic thrombocytes].
Khvoroba Glanzmann. Syndrome of syrich thrombocytes. Thrombastenia (hemorrhagic) (spadkov). Thrombocytopathy.
Vicked: Willebrand's ailment (D68.0)
D69.2 Insha non-thrombocytopenic purpura.
purpura:
... NOS
... senile
... simple
D69.3 Idiopathic thrombocytopenic purpura. Evans syndrome
D69.4Інші primary thrombocytopenia.
Viklyucheni: thrombocytopenia from the daytime exchange list(Q87.2)
past neonatal thrombocytopenia (P61.0)
Wiskott-Oldrich syndrome (D82.0)
D69.5 Secondary thrombocytopenia. If it is necessary to identify the cause of the vikoristovuyut additional code of the underlying reasons (class XX).
D69.6 thrombocytopenia, unspecified
D69.8Іnshi clarification of the hemorrhagic hospital. Capillary lamina (spadkov). Sudden pseudohemophilia
D69.9 Hemorrhagic camp of unspecified

INSHI BLOOD BLOOD OF BLOOD ORGANS (D70-D77)

D70 Agranulocytosis

Agranulocytic angina. Childish genetic agranulocytosis. Kostmann's ailment
neutropenia:
... NOS
... vrogena
... cyclic
... medication
... periodic
... splenic (pervinna)
... toxic
neutropenic splenomegaly
If necessary, identifying a lykarskiy zyb, who has experienced neutropenia, has a vicious code of common reasons (class XX).
Vicked: past neonatal neutropenia (P61.5)

D71 Functional destruction of polymorphonuclear neutrophils

Defect in the receptor complex of the cell membrane. Chronic (childish) granulomatosis. natural dysphagocytosis
Progressive septic granulomatosis

D72 Diseased blood cells

Viklyucheni: basophilia (D75.8)
immune damage (D80-D89)
neutropenia (D70)
preleukemia (syndrome) (D46.9)

D72.0 Genetic anomalies of leukocytes.
Anomaly (granulation) (granulocyte) or syndrome:
... Aldera
... May-Hegglina
... Pelger-Hueta
spadkova:
... leukocyte
... hypersegmentation
... hyposegmentation
... leukomelanopathy
Exclusion: Chediaka-Higashi (-Steinbrinka) syndrome (E70.3)
D72.1 Eosinophilia.
eosinophilia:
... allergic
... spadkova
D72.8Іnshi clarification of the destruction of many blood cells.
Leukemic reaction:
... lymphocytic
... monocytic
... mієlocytarna
Leukocytosis. Lymphocytosis (symptomatic). Lymphopenia. Monocytosis (symptomatic). Plasmacytosis
D72.9 Destruction of many blood cells of unspecified

D73 Spleen ailments

D73.0 Hyposplenism. Asplen_ya of pislyaoperats_yna. Atrophy of the spleen.
Vicked: asplenia (vrogena) (Q89.0)
D73.1 hypersplenism
Viklyucheni: splenomegaly:
... NOS (R16.1)
.vrojena (Q89.0)
D73.2 Chronic persistence of splenomegaly
D73.3 splenic abscess
D73.4 cyst of the spleen
D73.5 Splenic infarction. Cutting the spleen is non-traumatic. Twisting of the spleen.
Victory: traumatic opening of the spleen (S36.0)
D73.8Іnshі spleen ailments. Fibrosis of the spleen NOS. Perspective. spleno NOS
D73.9 Spleen ailment, unspecified

D74 Methemoglobinemia

D74.0 Vrodzhena methemoglobinemia. Lack of NADH-methemoglobine reductase is likely.
Hemoglobin M [ailment Hb-M]. Methemoglobinemia spadkov
D74.8Інші methemoglobinemia. Predbana methemoglobinemia (s sulfhemoglobinemia).
Methemoglobinemia is toxic. If it is necessary to identify the cause of the vikoristovuyut additional code of the underlying reasons (class XX).
D74.9 methemoglobinemia, unspecified

D75 Other ailments of the blood and hematopoietic organs

Viklyucheni: zbіlshennya lymphatic universities(R59. -)
hypergamaglobulinemia NOS (D89.2)
lymphadenitis:
... NOS (I88.9)
... gostry (L04 .-)
... chronic (I88.1)
... brijovikh (gostry) (chronic) (I88.0)

D75.0 Symeiniy erythrocytosis.
polycythemia:
... kindly
... simeina
Inclusion: decongestant ovalocytosis (D58.1)
D75.1 Secondary polycythemia.
polycythemia:
... dribble
... tied with:
... erythropoietins
... Decreased obsyachy plasmi
... hanging
... stress
... emotsiyna
... Hypoxemic
... nephrogenic
... obvious
Viklyucheni: polycythemia:
... newborn (P61.1)
... true (D45)
D75.2 Essential thrombocytosis.
Viklyuchena: esential (hemorrhagic) thrombocythemia (D47.3)
D75.8Іnshi clarification of ailments of the blood and blood-forming organs. basophilia
D75.9 Ailment of blood and blood-forming organs, unspecified

D76 Acute ailments that pass through the lesions of the lymphoretic tissue and the reticulohistiocytic system

Victims: Letterer-Siwe ailment (C96.0)
malignant histiocytosis (C96.1)
reticuloendotheliosis or reticulosis:
... histiocytic cerebral (C96.1)
... leukemic (C91.4)
... lipomelanotic (I89.8)
... malignant (C85.7)
... unfinished (C96.0)

D76.0 Histiocytosis from Langerhans' cells, not classified in the same headings. Eosinophilic granuloma.
Khvoroba hand-Schüller-Krisgen. Histiocytosis X (chronic)
D76.1 Hemophagocytic lymphocytosis. Family hemophagocytic reticulosis.
Histiocytosis from phagocytes, specific species of Langerhans' clitin, NOS
D76.2 Hemophagocytic syndrome, dressings with infection.
If necessary, the identification of an infectious agent or a disease can be identified as an additional code.
D76.3Інші hіstіocytosis syndromes. Retіkulogіstіocytoma (giant cell).
Sinus histiocytosis with massive lymphadenopathy. Xanthogranuloma

D77 Інші broken blood and hematopoietic organs with ailments, classified in the same headings.

Fibrosis of the spleen in schistosomiasis [bilharziasis] (B65. -)

SINCER DISTURBANCE due to the findings of the immune mechanism (D80-D89)

Included: defects in the complement system, immunodeficiency disorders, with a slight ailment,
viral infection of human immunodeficiency [VIL] sarcoidosis
Victims: autoimmune ailments (systemic) NOS (M35.9)
functional destruction of polymorphonuclear neutrophils (D71)
ailment, viclikana virus, human immunodeficiency virus [VIL] (B20-B24)

D80 Immunodeficiency due to severe lack of antibodies

D80.0 Spadkova hypogamaglobulinemia.
Autosomal recessive agammaglobulinemia (Swiss type).
X-chromosome bleeding agammaglobulinemia [Bruton] (growth hormone deficiency)
D80.1 There is no hypogammaglobulinemia. Agammaglobulin with the presence of B-lymphocytes, which are immunoglobulin. Zagalny agammaglobulinemia. hypogammaglobulinemia NOS
D80.2 Vibrating deficiency of immunoglobulin A
D80.3 Vibrating deficiency in immunoglobulin G
D80.4 Vibrating deficiency of immunoglobulin M
D80.5 Immunodeficiency with increased immunoglobulin M
D80.6 Lack of antibodies with close to normal levels of immunoglobulin or hyperimmunoglobulinemia.
Deficiency of antibodies with hyperimmunoglobulinemia
D80.7 Minusch hypogammaglobulinemia children
D80.8Іnshі immunodeficiency with a major defect in antibodies. Kappa-lung lantsyugiv deficiency
D80.9 Immunodeficiency with a major defect of antibodies, unspecified

D81 Combined immunodeficiency

Excludes: autosomal recessive agammaglobulinemia (Swiss type) (D80.0)

D81.0 An important combination of immunodeficiency with reticular dysgenesis
D81.1 An important combination of immunodeficiency with a low volume of T- and B-clitin
D81.2 An important combination of immunodeficiency with low or normal b-cell volume
D81.3 adenosine deaminase deficiency
D81.4 Neselof's syndrome
D81.5 purine nucleoside phosphorus lase deficiency
D81.6 Deficiency of molecules of class I of the head complex of histosumity. Syndrome of "naked lymphocytes"
D81.7 Deficiency of molecules of class II of the head complex of histosumity
D81.8Іnshі combined immunodeficiency. Deficiency of bio-dependent carboxy-lase
D81.9 Combinations of immunodeficiency, unspecified. Vazhke Combined Immunodeficiency Disorder NOS

D82 Immunodeficiency associated with significant defects

Invalid: atactic teleangiectasia [Luї-Bar] (G11.3)

D82.0 Wiskott-Aldrich syndrome. Immunodeficiency with thrombocytopenia and eczemoy
D82.1 Di Georg's syndrome. Pharyngeal diverticulum syndrome.
Vilochkovoi zalozi:
... alimphoplasia
... aplasia or hypoplasia with immune deficiency
D82.2 Immunodeficiency with dwarfism for short cakes
D82.3 Immunodeficiency due to a recessive defect caused by the Epstein-Barr virus.
X-chromosome blending of lymphoproliferative ailment
D82.4 Hyperimmunoglobulin E syndrome
D82.8 Immunodeficiency, dressings with additional clarifications of significant defects
D 82.9 Immunodeficiency, dressing with a significant defect, unspecified

D83 Serious variable immunodeficiency

D83.0 Zagalny variable immunodeficiency with major failures in the number and functional activity of B-clitin
D83.1 Zagalny variably immunodeficiency with overgrowths of immunoregulatory T-clitin
D83.2 Zagalny variable immunodeficiency with autoantibodies up to B- or T-cells
D83.8Інші local variable immunodeficiency
D83.9 Zagalny variable immunodeficiency, unspecified

D84 Іnshі immunodeficiency

D84.0 Defect of lymphocyte functional antigen-1
D84.1 A defect in the complement system. Deficiency of C1 angibitor esterasi
D84.8Іnshi clarification of immunodeficiency disorders
D84.9 immunodeficiency, unspecified

D86 Sarcoidosis

D86.0 sarcoidosis of legends
D86.1 Sarcoidosis of lymphatic universities
D86.2 Sarcoidosis of legends from sarcoidosis of lymphatic universities
D86.3 sarcoidosis of shkiri
D86.8 Sarcoidosis of other specified and combined localizations. Iridocyclitis with arterial disease (H22.1).
many parallels cranial nerves adipose (G53.2)
Sarcoid (s):
... arthropathy (M14.8)
... myocarditis (I41.8)
... myositis (M63.3)
Uveoparaotic fever [Herfordt's ailment]
D86.9 unspecified sarcoidosis

D89 Disorder due to the findings of the immune mechanism, not classified in other headings

Viklyucheni: hyperglobulinemia NOS (R77.1)
monoclonal gammapathy (D47.2)
neoplasm of transplantation (T86 .-)

D89.0 Polyclonal hypergamaglobulinemia. Hypergammaglobulinemic purpura. Polyclonal gammapathy NOS
D89.1 Cryoglobulinemia.
krioglobulinemia:
... essential
... idiopathic
... zmіshana
... pervinna
... second
Cryoglobulinemic (s):
... purpura
... vasculitis
D89.2 hypergammaglobulinemia, unspecified
D89.8Інші clarification of damage from the findings of the immune mechanism, not classified in the other headings
D89.9 Damage, scho zaluchaє immune mechanism, unspecified. Immunna ailment NOS

Have great people All the main values ​​of the blood are guilty of being in the norm, be it a vidhilennya - a sign of the development of pathological processes. Anemia is characterized by a decrease in the number of red blood cells and a low hemoglobin, which causes ailments to cause ailments or embarrassing character, and often becomes ill due to improper feeding.

Through the fast chervonykh clitin, they call anemia underwhelmed

Anemia - well tse take?

- ailments, yak manifest themselves in significant reductions in hemoglobin and in the number of blood hearts. The code for the engagement according to ICD-10 is D50-D89.

Anemia is not a headache, pathology is likely to develop on the basis of diseases in robots internal organs and systems.

Classification of anemia

Oscillations of the reasons for the development of anemia are rich, the stench is manifested by small symptoms, the skin form of vimag is especially drug therapy, Zahvoryuvannya klasifіkuyut for singing indicators.

In any form of anemia, the value of hemoglobin is lower than the permissible values, and the number of erythrocytes can be normal or decrease.

For a colonic indicator

Color indicator- Rivn saturation of red blood cells with hemoglobin. For the development of the erythrocyte index, the hemoglobin should be multiplied by 3, and the number of erythrocytes should be multiplied by the whole.

Classification:

With normochromic anemia, the indicators should not go beyond the permissible limits

• hypochromic- color index up to 0.8 units;
• normochromic- color index 0.6-1.05 units;
• hyperchromic- the value of the colonic indicator perevischuє 1.05 units.

The diameter of the erythrocytes is 7.2-8 microns. The change in size is a sign of the deficiency of vitamins B-9, B-12, the change is made about the marriage.

For the growth of the cystic brain before regeneration

The process of the establishment of new cells is seen in the tissues of the main organ of the hematopoietic system, the main indicator of normal robotic organism is the presence in the blood of the required number of reticulocytes, primordial worm cells, the ability of these cells to be called heritrophy.

Classification:

• Regenerative - the number of reticulocytes is 0.5-2%, the rate of regeneration is normal;
• hiporegenerative - to promote a decrease in regenerative functions, the number of reticulocytes is 0.5%;
• overregenerative - acceleration process renewal of tissue in the cystic marrow, reticulocytes in the blood is more than 2%;
• aplastic - reticulocytes are visible, or even 0.2%.

It takes 2-3 years to synthesize new red blood cells.

For the mechanism of development of pathology

The anemia of the blood loss due to the destruction of the process of the formation of the hearts of the blood or of the violent destruction, for the mechanism of the development of the ailment go to the list of categories.

see:

• anemia through severe blood loss, chronic bleeding;
• zalizodeficitna, renal, B12 and folate form, aplastic - ailments are diagnosed through problems in the process of blood formation;
• at deyakikh autoimmune vidhilennyah, on the nasty slump it is possible to ruin erythrocytes, develop anemia.

Short-hour anemia of the easy step of the vine in women before the hour of the month, when they are flat. Even in the body, there are no serious vidhilen, for the improvement of self-esteem to complete the speedy race, normalize the regime of the day.

Grades of severity of anemia

There are 3 stages of the severity of the pathological camp, in the presence of the fact that the hemoglobin value is actually lower than the permissible norm.

normal hemoglobin

Persh nіzh classify anemia to carry out an analysis for the level of hemoglobin

Grades of severity:

• 1 step - hemoglobin in between 90 g / l;
• 2 steps - hemoglobin 70-90 g / l;
• 3 steps - hemoglobin 70 g / l or less.

Light forms of becoming ill are characterized by insignificant deterioration of the body, the importance of anemia does not pose a serious threat to the health of grown-up people, for a child pathological changes can be deadly.

Symptoms and clinical manifestations

In case of anemia, the gas exchange breaks down, for a decrease in the number of erythrocytes, it is more difficult to transport gas and kissen in carbon dioxide. One of the main signs of any kind of ailment is anemic syndrome - an attack of foulness, drowsiness, sickness, urgency, bliss, headache. Photos of ailments of people to allow the appearance of signs of ailments.

Anemia, scho vinikla through erosive gastritis

In case of anemia, spite the blida shkira

When there is a lack of balance, wonderful relish addictions appear - people want to eat meat, sire meat. Spostering and smelling things - we have a problem to be like the smell of barvniks, gasoline.

Causes of failure

Anemias are a legacy of mass or trivial bleeding, a decrease in the speed of the appearance of new erythrocytes, a violent ruin of hearts of blood. Ailments are often indicative of chronic abnormal deficiency of saline, folate and ascorbic acid, vitamin B12, with overwhelmed suvorim children, hunger.

The twisting of the drain can be the cause of chronic blood loss

Pseudoanemia - a change in the viscosity of the blood when there is an increase, through the overworld of birth. Anemia - thickening of blood, winy with ryevyuvuot, diarrhea, increased drinkability, indicators of hemoglobin and erythrocytes do not decrease.

In some cases, people diagnose anemia, a decrease in hemoglobin of an unclear genesis, if the exact cause is found, or one reason for the diagnosis of pathology is not to go to the problem of realizing.

A decrease in hemoglobin in children often has a natural character, second anemia- the legacy of unbalanced eating, active growth in puberty.

Thalassemia is an important part of the disease, the diagnosis is made through the adjustment of the disease to the hemoglobin, the erythrocytes are in the form of the target. Signs - zhivtushn_st, earthy-greenery of the shkіrnyh curves, the irregular shape of the skull and the ruin of Budovi knitted fabric, Vidhilennya in pink, physical development, Ochi Mayut Mongolian growth, oven and spleen improved.

The main signs of anemia are zhivtushnist and bilizna

Hemolithic anemia of new women- vinikє through a rhesus-conflict, a child with a population diagnosed with strong swellings, ascites, in the blood of a lot of immature erythrocytes. The stage of pathology begins on the basis of indicators of hemoglobin and indirect bilirubin.

Spherocytic - decay of the gene pathology, with yaky erythrocytes may be rounded, swiftly ruinous in the selenium. Heritage - setting stones in zhovchny mikhury, Zhovtyanitsya, gratitude, nervousness.

Until what lykary will you turn around?

In case of manifestations of anemia, it is necessary for the ear. In order to reject the results of the primary diagnostics, the present patients will be busy. With a suspicion of the appearance of internal bleeding, pukhlin is required for a hospital term.

diagnostics

The main type of diagnosis- flare-up and secondary blood analysis, with the help of a hematological analyzer, there is a number of erythrocytes, especially of their buds, the value of a colonic indicator, hemoglobin, detection of ignition processes.

To identify pathology, create a full range of blood analyzes

Diagnostic methods:

• biochemistry of blood;
• analysis of the section for hemoglobin detection;
• doslіdzhennya feces for the appearance of adherent blood, yєts worms;
• fibrogastroduodenoscopy, colonoscopy - assessment of the shlunka and other organs in the shlunkovo-intestinal tract;
• mylogram;
• Ultrasound examination of organs of the reproductive, herbal, dichal systems;
• CT legends, nirok;
• fluorography;
• EKG, echocardiography;

Erythrocytes in the middle live for 90-120 days, with a drop (hemolysis) occur in the middle of the blood cells, in the cystic brain, liver and blood cells. Be-like troubles in robotic organs provoke anemia.

lykuvannya anemia

To increase the hemoglobin level, the preparations are in tablet form, in the form of tablets for those who are used to, droplets, as they learn the main cause of anemia, to do something like folk methods.

When diagnosing internal bleeding, perform an operation, in important cases, transfusion or purification of blood is required, transplantation of the cystic marrow, and spleen vision.

liks

Drugs are selected on the basis of indicators of the results of analyzes, the type and degree of severity of anemia, the main diagnosis.

Chim Likuvati:

Aktiferrin - iron supplement

• Aktiferrin, Ferlatum - preparations of zaliza, priznachayut in a complex with vitamin C;
• internal introduction of Vitamin B12;
• liquor with folin acid;
• immunodepression, antimetabolity - Methodject, Ekoral;
• glucocorticosteroids - Prednizol, Medopred;
• rіznі vidi immunoglobulinіv;
• Zasobi for accelerating the process of setting up erythrocytes in stovburovikh cells - Epotal, Vepox.

In case of strong blood loss, go in for a replenishment of the amount of circulating blood - for an additional drip, inject erithrocytic mass, albumin, Polyglyukin, gelatinol, glucose.

People zasobi

methods unconventional medicine normalize the values ​​of the main indicators of blood in mild forms of anemia, with important, chronic diseases of ailments and stagnation only in the form of pre-treatment therapy given in the course of an early consultation with a doctor.

Simple recipes:

1. Zmіshati in equal proportions of black radish, carrot, beetroot, tomiti sumish in the spirits shafі for the minimum period of 3 years. Dose for older adults - 15 ml, for children - 5 ml, take a few drinks for doba.
2. Add 100 g of fresh polin, pour 1 liter of the bottle, clean in the dark for 21 days. Vzhivat 5 drops before the cutaneous reception of the їzhі.
3. Up to 200 ml of pomegranate juice dodati, 100 ml of carrot, apple and lemon juice, 70 ml of wild honey. Put sumish in the refrigerator for 48 years. Piti 30 ml three for doba.
4. Podrіbnity 300 g of peeled chalk, pour 1 liter of the bottle, tidy up in the dark for 3 days. Drink 5 ml in front of the drink.
5. Take 175 ml of aloe juice, 75 ml of honey and 450 ml of Cahors, put it back in the refrigerator. Drink 30 ml of three on the day before the meal.

The simplest method for usunennya and prevention of anemia is to regularly vzhivat nastiy s yagid shipshini, 1 tbsp. l. brew 1 liter of sprinkle, insist 8 years in a thermos, for good wrapping up the castruli.

In case of poor forms of anemia during the season, live 2 kg of kavuna, there is no evidence of opposition.

May inheritance and acceleration

Without the right and timely therapy, the anemia is signi fi cantly weak immune system, To improve the development of important viral and bacterial pathologies.

Chim is not without preservation:

• Legeneva, nirkova and heart failure;
• becoming ill with a neurological character;
• destruction of memory, concentration of respect;
• deformation of shkiri, mucous membranes;
• vidhilennya in pink and physical development in children;
• chronic ailments of the eyes, organs of the herbal and dichal systems.

One of the legacies of anemia - the destruction of memory

With important forms of incomplete tissue, tissue hypoxia develops, which can be the cause of hemorrhagic and cardiogenic shock, hypotonia, coma, and a lethal result.

Features of anemia with vaginosis

All vaginities enter the group, anemia during the whole period is often diagnosed, or hemoglobin indicators and the number of erythrocytes decrease slightly, the country is normal. cause- Decrease of rare warehouse blood for a decrease in blood supply.

In some cases of frequent bloating with toxicosis, in case of problems with wetness of the disease, anemia, pathology is more likely to develop in case of two or more children, with frequent vaginities.

symptoms- vtoma, weakness, sleeplessness or drowsiness, strong offense, Nudota, to the point of obscurity. Shkira grows dry and bleed, nigty lamayutsya, hairs strongly vipadaє. Such a camp can be the cause of a wicked day, preeclampsia, a pre-hour rozrozhennya, the curtains are inviting to pass vigorously. In vaginal lower boundaries, hemoglobin levels are 110 mg / l.

basis of therapy- day, in the menu it is guilty that there are more offal, dietary meat, ribi, in addition, 15-35 mg of saline is needed, in the presence of the term of vaginosity. Dodatkovo priznachayut preparations with ascorbic and folic acid, sulfate and hydroxide of zaliz.

If the woman was diagnosed with anemia before the hour of vaginosity, then often there is a deficiency in the health of the child in the first time of life.

prophylaxis

Decrease of anemia's definition of anemia is more correct, an unbalanced meal is a change in the living of food fats, replacing them with growing ones, unique children, living more honey, Greek and porridge, Vegetables, fruits, berries.

Regular sports to keep your shelter and keep it practical if you have ailments

All kinds of liver, yalovic language, yalovichina and poultry meat, riba, peas, greek porridge, beetroot, cherries and apples - all of the products are rich in zalizom;

- broader incapacitation, women develop 10 times more often, less than choloviks. Suchasnі lіkarskі zasobi, folk recipes effectively help to fit in with the pathology, uniqueness the acceleration, and after simple visits to prophylaxis - to reduce the risk of developing ailments.

ICD 10. Class III. Ailments of blood, hematopoietic organs, and the surrounding of damage to the results of the immune mechanism (D50-D89)

Victims: autoimmune ailments (systemic) NOS (M35.9), near the village, occurring at the perinatal period (P00-P96), accelerated vaginosity, lower and lower periods of hypoplasia (O00-O99), deformity - Q99), endocrine ailments, problems with the disease and destruction of the disease (E00-E90), ailment, sickness, infection with the virus of human immunodeficiency [VIL] (B20-B24), injuries, rejection of these causes for reasons new findings (C00-D48), symptoms, signs and symptoms of failure according to norms, revealed in clinical and laboratory diagnostics, not classified in lower headings (R00-R99)

Tsey class to take revenge on the offensive blocks:

D50-D53 Anemias, knitted from food

D55-D59 Hemolytic anemias

D60-D64 Aplastic and insignificant anemias

D65-D69 Loss of laryngeal blood, purpura and other hemorrhagic beds

D70-D77 Other ailments of the blood and hematopoietic organs

D80-D89 Damage caused by the immune mechanism

The third category is as follows:

D77 Infringement of blood and hematopoietic organs with ailments classified in the same headings

ANEMI, CONNECTED TO LIVES (D50-D53)

D50 Zalizodeficitna anemia

D50.0 Zalizodeficitny anemia is secondary in cases of blood loss (chronic). Post-hemorrhagic (chronic) anemia.

Wikipedia: posthemorrhagic anemia (D62) congenital anemia during fetal hemorrhage (P61.3)

D50.1 Syderopenic dysphagia. Kelli-Paterson syndrome. Plummer-Winson syndrome

D50.8 Indigestion anemia

D50.9 Zalizodeficiency anemia, unspecified

D51 Vitamin-B12 deficiency anemia

Victims: Vitamin B12 deficiency (E53.8)

D51.0 Vitamin-B12 deficiency anemia due to internal factor deficiency.

There is a lack of an internal official

D51.1 Vitamin-B12 deficiency anemia due to vibrational damage due to wetting Vitamin B12 with protein.

Imerslund (-Gresbeck) syndrome. Megaloblastic spadkov anemia

D51.2 Transcobalamin II deficiency

D51.3 Inshi vitamins-B12-deficient anemias, associated with food. anemia of vegetarians

D51.8 Inshi vitamins-B12-deficient anemias

D51.9 Vitamin-B12 deficiency anemia, unspecified

D52 Folio-water deficiency anemia

D52.0 Folio-water deficiency anemia, associated with abnormalities. Megaloblastic alimentary anemia

D52.1 Folio-water deficiency anemia is medication. If necessary, identify the license.

vikoristovuyt additional code of calling reasons (class XX)

D52.8 Follicle water deficiency anemia

D52.9 Foliate deficiency anemia, unspecified Anemia caused by the lack of proper functioning in the organism of folic acid, NOS

D53 Inshi anemia, knitted with a crochet

Included: megaloblastic anemia,

nom B12 abo folate

D53.0 Anemia due to deficiency of blood cells. Anemia due to lack of amino acids.

Viklyuchenii: Lesch-Nihen syndrome (E79.1)

D53.1 Some megaloblastic anemias, not classified in any of the other headings. Megaloblastic anemia NOS.

Excluded: ailment Di Gul'lmo (C94.0)

D53.2 Anemia, surrounded by scurvy.

Vicious: scurvy (E54)

D53.8 Other related anemia related to food.

Anemia, associated with a deficiency:

Viklyuchenya: lack of food without a puzzle about

anemia, such as:

Deficiency of midi (E61.0)

Molybdenum deficiency (E61.5)

Zinc deficiency (E60)

D53.9 Anemia, associated with food, unspecified. Simple chronic anemia.

Excluded: anemia NOS (D64.9)

Hemolytic anemia (D55-D59)

D55 Anemia due to enzyme damage

Excluded: enzyme deficiency anemia, vicylican lyceum diseases (D59.2)

D55.0 Anemia due to deficiency of glucose-6-phosphate dehydrogenase [G-6-PD]. Favism. G-6-PD-deficiency anemia

D55.1 Anemia due to damage to glutathione metabolism.

Anemia due to lack of enzymes (with the guilt of G-6-PD), tied from hexose monophosphate [HMP]

shunt metabolic path. Hemolytic non-spherocytic anemia (spadkov) type 1

D55.2 Anemia due to damage to glycolytic enzymes.

Hemolytic non-spherocytic (spadkov) type II

Due to the lack of hexokinase

Due to the lack of pyruvate

Due to the lack of triose phosphate isomerase

D55.3 Anemia due to impairment of nucleotide metabolism

D55.8 Anemia due to enzyme damage

D55.9 Anemia due to enzyme damage, unspecified

D56 Thalassemia

Fetal dropsy, surrounded by hemolytic ailments (P56. -)

D56.1 Beta thalassemia. Anemia Kuli. Beta-talasemia is important. Sickle-cell beta-thalassemia.

D56.3 Nose signs of thalassemia

D56.4 Spasmodic persistence of fetal hemoglobin [APFH]

D56.9 Thalassemia, unspecified Seredzemomorska anemia (s)

Thalassemia (mala) (zmіshana) (from the least hemoglobinopathy)

D57 sickle-shaped disease

Viklyucheni: іnshі hemoglobinopathies (D58. -)

sickle-clitinna beta-thalassemia (D56.1)

D57.0 sickle-cell anemia with crisis. Hb-SS ailment with crisis

D57.1 sickle-cell anemia without crisis.

D57.2 Subclinical heterozygous sickle cell disease

D57.3 Nosystvo mark of sickle-clitinous. Nose to hemoglobin S. Heterozygous hemoglobin S

D57.8 - Crescent-shaped disease

D58 Lowest hemolytic anemia

D58.0 Spherocytosis spherocytosis Akholurichna (simeina) zhovtyanytsya.

Vrodzhena (spherocytic) hemolytic zhovtyanitsa. Mankivsky-Shoffard syndrome

D58.1 Subsidious elliptocytosis. Ellitotsitosis (congenital). Ovalocytosis (congenital) (decline)

D58.2 Inshi hemoglobinopathies. Abnormal hemoglobin NOS. Anemia with Heinz's children is born.

Hemolytic ailment, zoomed with unstable hemoglobin. Hemoglobinopathy NOS.

Viklyucheni: familyna polycythemia (D75.0)

ailment Hb-M (D74.0)

decline in persistence of fetal hemoglobin (D56.4)

polycythemia, tied with visota (D75.1)

D58.8 Inspection of decrease in hemolytic anemia. stomatocytosis

D58.9 Spadkova hemolytic anemia, unspecified

D59 Nabuta hemolytic anemia

D59.0 Medication autoimmune hemolytic anemia.

If necessary, identify the drug as a vicorist for the additional code of reasons (class XX).

D59.1 Inshi autoimmune hemolytic anemias. Autoimmune hemolytic ailment (cold type) (heat type). Chronic ailments

Cold type (secondary) (symptomatic)

Thermal type (secondary) (symptomatic)

Victims: Evans syndrome (D69.3)

hemolytic ailment of fetus and neonatal (P55. -)

paroxysmal cold hemoglobinuria (D59.6)

D59.2 Medication non-autoimmune hemolytic anemia. Enzyme-deficient anemia medication.

If necessary, identifying a likarskiy zakorystovuyut an additional code of specific reasons (class XX).

D59.3 hemolytic uremic syndrome

D59.4 Non-autoimmune hemolytic anemias.

If it is necessary to identify the cause of the vikoristovuyut additional code of the underlying reasons (class XX).

D59.5 Paroxysmal non-hemoglobinuria [Markiafavi-Mikele].

D59.6 Hemoglobinuria due to hemolysis due to other underlying causes.

Excludes: hemoglobinuria NOS (R82.3)

D59.8 Other hemolytic anemias

D59.9 Abnormal hemolytic anemia, unspecified. Idiopathic hemolytic anemia is chronic

D60 Nabuta pure red cell aplasia (erithroblastopenia)

Included: red cell aplasia (dribbana) (grown-up) (with Tim)

D60.0 Chronic dribbling pure red cell aplasia

D60.1 Timchasova dribble pure red cell aplasia

D60.8 Inshі nabutі pure red cell aplasia

D60.9 Pridbana pure red cell aplasia, unspecified

D61 Aplastic anemias

Exclusion: agranulocytosis (D70)

D61.0 Constitutional aplastic anemia.

Aplasia (pure) red cell:

Blackfen-Daymond syndrome. Semeina is hypoplastic anemia. Anemia Fanconi. Pancytopenia with vads development

D61.1 Drug-induced aplastic anemia. If necessary, identify the license.

vikoristovuyt additional code of calling reasons (class XX).

D61.2 Aplastic anemia caused by other calling agents.

If it is necessary to identify the cause of the vikoristovuyut additional code of the underlying reasons (class XX).

D61.3 Idiopathic aplastic anemia

D61.8 Other refined aplastic anemias

D61.9 Aplastic anemia, unspecified Hypoplastic anemia NOS. Hypoplasia of the cystic marrow. panmieloftiz

D62 Gostra post-hemorrhagic anemia

Excluded: congenital anemia due to fetal haemorrhage (P61.3)

D63 Anemia with chronic ailments, classified in the same headings

D63.0 Anemia with new conditions (C00-D48 +)

D63.8 Anemia with other chronic ailments, classified in the same headings

D64 Інші anemia

Too blast (D46.2)

Transformation (D46.3)

3 sideroblasts (D46.1)

Without sideroblasts (D46.0)

D64.0 Spadkova sideroblastic anemia. Hypochromic sideroblastic anemia is associated with the disease

D64.1 Secondary sideroblastic anemia associated with others.

If necessary, identifying a pre-existing code.

D64.2 Secondary sideroblastic anemia, viclican with drugs and toxins.

If it is necessary to identify the cause of the vikoristovuyut additional code of the underlying reasons (class XX).

D64.3 Inshi sideroblastic anemia.

Peridoxin-responsive, not classified in these headings

D64.4 Disease-tropoietic anemia is susceptible to disease. Dyshemopoietic anemia (born).

Vicious: Blackfen-Daymond syndrome (D61.0)

ailment Di Gul'lmo (C94.0)

D64.8 Inspection of anemia. Childish pseudo leukemia. Leukoeritroblastic anemia

Destruction of the throat of blood, purpura TA INSHI

Hemorrhagic STANE (D65-D69)

D65 Dissemination of the internal vascular laryngeal [defibrillation syndrome]

Afibrinogenemia pridebana. coagulopathy

Diffuse or disseminated internally

Fibrinolytic bleeding of the dribble

Vklyucheniy: syndrome defibrіnatsіі (scho accelerated):

Newborn (P60)

D66 Substitious factor VIII deficiency

Factor VIII deficiency (with functional impairment)

Inclusion: Factor VIII deficiency in vascular lesions (D68.0)

D67 Subsidious factor IX deficiency

Factor IX (with functional impairment)

Thromboplastic component of plasmas

D68 Disease of the throat

Abortion, post-mortem or molar vaginism (O00-O07, O08.1)

Viability, canopy and non-linear period (O45.0, O46.0, O67.0, O72.3)

D68.0 Villebrand's ailment. Angiohemophilia. Deficiency of factor VIII in cadaverous lesions. Sudden hemophilia.

Victory: Spadkov capillary lameness (D69.8)

factor VIII deficiency:

Functional damage (D66)

D68.1 Subsidious factor XI deficiency. Hemophilia C. Deficiency of thromboplastin transplanted plasma

D68.2 Subsidious deficiency of other factors of the larynx. Vrodzhena afіbrinogenemia.

Disfibrinogenemia (vrodzhena). Ovren's ailment

D68.3 Hemorrhagic damage caused by circulating anticoagulants. Hyperheparinemia.

If necessary, identification of vikoris anticoagulant vikoristovyt additional code of reasons

D68.4 Laryngeal factor deficiency.

Laryngeal factor deficiency for some reason:

Disadvantages of Vitamin K

Excluded: lack of vitamin K in a newborn (P53)

D68.8 Inspection of laryngeal failure. The presence of the ingibitor of the systemic heart

D68.9 Loss of throat, unspecified

D69 Purpura and other hemorrhagic beds

Viklyucheni: good hypergammaglobulinemic purpura (D89.0)

cryoglobulinemic purpura (D89.1)

idiopathic (hemorrhagic) thrombocythemia (D47.3)

bliscavic purpura (D65)

thrombotic thrombocytopenic purpura (M31.1)

D69.0 Allergic purpura.

D69.1 Defects in platelets. Bernard-Sul'e syndrome [hypertrophic thrombocytes].

Khvoroba Glanzmann. Syndrome of syrich thrombocytes. Thrombastenia (hemorrhagic) (spadkov). Thrombocytopathy.

Vicked: Willebrand's ailment (D68.0)

D69.2 Insha non-thrombocytopenic purpura.

D69.3 Idiopathic thrombocytopenic purpura Evans syndrome

D69.4 Primary thrombocytopenia.

Viklyucheni: thrombocytopenia from the outside of the promenade (Q87.2)

past neonatal thrombocytopenia (P61.0)

Wiskott-Oldrich syndrome (D82.0)

D69.5 Secondary thrombocytopenia. If it is necessary to identify the cause of the vikoristovuyut additional code of the underlying reasons (class XX).

D69.6 Thrombocytopenia, unspecified

D69.8 Inspection of hemorrhagic disease. Capillary lamina (spadkov). Sudden pseudohemophilia

D69.9 Hemorrhagic camp of unspecified

INSHI BLOOD BLOOD OF BLOOD ORGANS (D70-D77)

D70 Agranulocytosis

Agranulocytic angina. Childish genetic agranulocytosis. Kostmann's ailment

If necessary, identifying a lykarskiy zyb, who has experienced neutropenia, has a vicious code of common reasons (class XX).

Vicked: past neonatal neutropenia (P61.5)

D71 Functional destruction of polymorphonuclear neutrophils

Defect in the receptor complex of the cell membrane. Chronic (childish) granulomatosis. natural dysphagocytosis

Progressive septic granulomatosis

D72 Diseased blood cells

Viklyucheni: basophilia (D75.8)

immune damage (D80-D89)

preleukemia (syndrome) (D46.9)

D72.0 Genetic abnormalities of leukocytes.

Anomaly (granulation) (granulocyte) or syndrome:

Exclusion: Chediaka-Higashi (-Steinbrinka) syndrome (E70.3)

D72.8 Inspection of damage to many blood cells.

Leukocytosis. Lymphocytosis (symptomatic). Lymphopenia. Monocytosis (symptomatic). Plasmacytosis

D72.9 Damage of blood cells, unspecified

D73 Spleen ailments

D73.0 Hyposplenism. Asplen_ya of pislyaoperats_yna. Atrophy of the spleen.

Vicked: asplenia (vrogena) (Q89.0)

D73.2 Chronic persistence of splenomegaly

D73.5 Splenic infarction. Cutting the spleen is non-traumatic. Twisting of the spleen.

Victory: traumatic opening of the spleen (S36.0)

D73.8 Other ailments of the spleen. Fibrosis of the spleen NOS. Perspective. spleno NOS

D73.9 Spleen ailment, unspecified

D74 Methemoglobinemia

D74.0 Congenital methemoglobinemia. Lack of NADH-methemoglobine reductase is likely.

Hemoglobin M [ailment Hb-M]. Methemoglobinemia spadkov

D74.8 Inshi methemoglobinemia. Predbana methemoglobinemia (s sulfhemoglobinemia).

Methemoglobinemia is toxic. If it is necessary to identify the cause of the vikoristovuyut additional code of the underlying reasons (class XX).

D74.9 Methemoglobinemia, unspecified

D75 Other ailments of the blood and hematopoietic organs

Viklyucheni: improvement of lymph universities (R59. -)

hypergamaglobulinemia NOS (D89.2)

Brizhovikh (gostry) (chronicle) (I88.0)

Inclusion: decongestant ovalocytosis (D58.1)

D75.1 Secondary polycythemia.

Decreased obscenity of plasmas

D75.2 Essential thrombocytosis.

Viklyuchena: esential (hemorrhagic) thrombocythemia (D47.3)

D75.8 Inspection of ailments of the blood and hematopoietic organs. basophilia

D75.9 Syndrome of blood and hematopoietic organs, unspecified

D76 Acute ailments that pass through the lesions of the lymphoretic tissue and the reticulohistiocytic system

Victims: Letterer-Siwe ailment (C96.0)

malignant histiocytosis (C96.1)

reticuloendotheliosis or reticulosis:

Histiocytic cerebral (C96.1)

D76.0 Hystiocytosis from Langerhans' clitin, not classified in these headings. Eosinophilic granuloma.

Khvoroba hand-Schüller-Krisgen. Histiocytosis X (chronic)

D76.1 Hemophagocytic lymphocytosis Family hemophagocytic reticulosis.

Histiocytosis from phagocytes, specific species of Langerhans' clitin, NOS

D76.2 Hemophagocytic syndrome, dressings with infection.

If necessary, the identification of an infectious agent or a disease can be identified as an additional code.

D76.3 Insidious syndromes Retіkulogіstіocytoma (giant cell).

Sinus histiocytosis with massive lymphadenopathy. Xanthogranuloma

D77 Інші broken blood and hematopoietic organs with ailments, classified in the same headings.

Fibrosis of the spleen in schistosomiasis [bilharziasis] (B65. -)

SINCER DISTURBANCE due to the findings of the immune mechanism (D80-D89)

Included: defects in the complement system, immunodeficiency disorders, with a slight ailment,

viral infection of human immunodeficiency [VIL] sarcoidosis

Victims: autoimmune ailments (systemic) NOS (M35.9)

functional destruction of polymorphonuclear neutrophils (D71)

ailment, viclikana virus, human immunodeficiency virus [VIL] (B20-B24)

D80 Immunodeficiency due to severe lack of antibodies

D80.0 Spadkova hypogamaglobulinemia.

Autosomal recessive agammaglobulinemia (Swiss type).

X-chromosome bleeding agammaglobulinemia [Bruton] (growth hormone deficiency)

D80.1 Non-essential hypogammaglobulinemia. Agammaglobulin with the presence of B-lymphocytes, which are immunoglobulin. Zagalny agammaglobulinemia. hypogammaglobulinemia NOS

D80.2 Vibrating deficiency of immunoglobulin A

D80.3 Vibrating deficiency in immunoglobulin G

D80.4 Vibrating deficiency of immunoglobulin M

D80.5 Immunodeficiency with increased immunoglobulin M

D80.6 Lack of antibodies for close to normal immunoglobulin or hyperimmunoglobulinemia.

Deficiency of antibodies with hyperimmunoglobulinemia

D80.7 Timchasov hypogammaglobulinemia of children

D80.8 Insidious immunodeficiency with major antibody defect. Kappa-lung lantsyugiv deficiency

D80.9 Immunodeficiency with major defect in antibodies, unspecified

D81 Combined immunodeficiency

Excludes: autosomal recessive agammaglobulinemia (Swiss type) (D80.0)

D81.0 Severe combination of immunodeficiency with reticular dysgenesis

D81.1 Severe combination of immunodeficiency with low volume of T- and B-clitin

D81.2 Severe combination of immunodeficiency with low or normal volume of B-cell

D81.3 Adenosine deaminase deficiency

D81.5 Purine nucleoside phosphorus lase deficiency

D81.6 Molecule deficiency in class I head gistosumnosty complex. Syndrome of "naked lymphocytes"

D81.7 Deficiency of molecules of class II of the head complex of histosumity

D81.8 Some combination of immunodeficiency. Deficiency of bio-dependent carboxy-lase

D81.9 Combinations of immunodeficiency, unspecified. Vazhke Combined Immunodeficiency Disorder NOS

D82 Immunodeficiency associated with significant defects

Invalid: atactic teleangiectasia [Luї-Bar] (G11.3)

D82.0 Wiskott-Aldrich syndrome. Immunodeficiency with thrombocytopenia and eczemoy

D82.1 Di Georg's syndrome. Pharyngeal diverticulum syndrome.

Aplasia or hypoplasia with immune deficiency

D82.2 Immunodeficiency with dwarfism for the development of short images

D82.3 Immunodeficiency due to a recessive defect caused by the Epstein-Barr virus.

X-chromosome blending of lymphoproliferative ailment

D82.4 Hyperimmunoglobulin E syndrome

D82.8 Immunodeficiency, dressings with further clarifications of significant defects

D82.9 Immunodeficiency, dressing with significant defect, unspecified

D83 Serious variable immunodeficiency

D83.0 General variable immunodeficiency with major failures in number and functional activity B-clitin

D83.1 General variable immunodeficiency with overgrowths of immunoregulatory T-cells

D83.2 Zagalny variable immunodeficiency with autoantibodies to B- or T-cells

D83.8 Indirect variable immunodeficiency

D83.9 External variable immunodeficiency, unspecified

D84 Іnshі immunodeficiency

D84.0 Defect of lymphocyte functional antigen-1

D84.1 Defect in complement system. Deficiency of C1 angibitor esterasi

D84.8 Inspection of immunodeficiency disorders

D84.9 Immunodeficiency, unspecified

D86 Sarcoidosis

D86.1 Sarcoidosis of lymphatic universities

D86.2 Sarcoidosis of lesions from sarcoidosis of lymphatic universities

D86.8 Sarcoidosis of other specific and combined localizations. Iridocyclitis with arterial disease (H22.1).

Multiple parallel cranial nerves in sarcoidosis (G53.2)

Uveoparaotic fever [Herfordt's ailment]

D86.9 Sarcoidosis, unspecified

D89 Disorder due to the findings of the immune mechanism, not classified in other headings

Viklyucheni: hyperglobulinemia NOS (R77.1)

monoclonal gammapathy (D47.2)

neoplasm of transplantation (T86 .-)

D89.0 Polyclonal hypergamaglobulinemia. Hypergammaglobulinemic purpura. Polyclonal gammapathy NOS

D89.2 Hypergammaglobulinemia, unspecified

D89.8 Insights into the findings of the immune mechanism, not classified in the other headings

D89.9 Damage due to immune mechanism, unspecified. Immunna ailment NOS

Aplastic TA INSHI ANEMIS (D60-D64)

Viklyucheni: refractory anemia:

• NOS (D46.4)
• too much blast (D46.2)
• transformation (C92.0)
• with sideroblasts (D46.1)
• without sideroblasts (D46.0)

The Russian International Classification of ailments of the 10th glance (ICD-10) has adopted a single normative document for the incidence of illness, the reasons for the infestation of the population in medical mortgage of all households, causes of death.

ICD-10 was introduced into the practice of protecting health in the entire territory of the Russian Federation in 1999 by the order of the Ministry of Health of Ukraine dated 27.05.97. No. 170

A new look (ICD-11) is planned by WHO in 2017 2018 roci.

From the changes and additions of the VOOZ rr.

Processing and transfer zmin © mkb-10.com

posthemorrhagic anemia

Post-haemorrhagic anemia is a sign of becoming ill, as it leads to a decrease in the number of erythrocytes and hemoglobin concentration through massive bleeding, or as a result of insignificant, albeit chronic, blood losses.

Hemoglobin is a blood complex of the erythrocyte, up to the storehouse where it can be entered. The main function of the field in the transfer of blood with the flow of blood to all without a blame for organs and tissues. In case of a disrupted process in the body, one tries to endure serious changes, as it starts with an ethological stage of the severity of anemia.

Falsely from the first reasons and the course of post-hemorrhagic anemias develop the state standard and chronic form. In relation to the system of international classification, they are given the following rank:

• Secondary hypersensitivity anemia due to blood loss. ICD code 10 D.50
• Gostra post-hemorrhagic anemia. Code for MKX 10 D.62.
• Congenital anemia due to bleeding in the fetus - P61.3.

V CLINIC PRACTICE Secondary hypertensive anemia is also called posthemorrhagic chronic anemia.

Reasons for the guest's form

The main reason for the development of post-hemorrhagic anemia is the loss of a great amount of blood in a short amount of time, as it was caused by:

• Traumas, yaka wikklikala pozhkogennya magistralnykh arterіy.
• Posthkodzhennya of the great blood-bearing judges from the hour of surgical engagement.
• Rise of the uterine tube with the development of post-vaginal vaginosity.
• Sickness of internal organs (most often lungs, nirok, heart, shlunkovo-intestinal tract), which can lead to massive internal bleeding.

In young children, the most frequent causes of post-hemorrhagic anemia are umbilical cord injuries, congenital pathology of the blood system, loss of the placenta in the course of caesarean roste, early placental discharges, overdue, birth trauma.

Causes of chronic interruption of post-hemorrhagic anemia

Chronic posthemorrhagic anemia develops as a result of minor, more regular bleeding. The stench can be found in the result:

• Hemorrhoids, which are supervised by rectal glands, will create houses of blood in the feces.
• Vibrant ailments of the slunk and twelve-fingered intestines.
• Rasnyh menstruation, uterine bleeding on the basis of hormonal drugs.
• Exercise of the Sudin with chubby clitines.
• Chronic nasal bleeding.
• Insignificant chronic blood loss in case of oncological illnesses.
• Parts of blood sampling, installation of catheters and other related procedures.
• It is important to prevent illnesses from seeing blood from the cross.
• Invasion by helminths.
• Liver cirrhosis, chronic liver failure.

The cause of chronic anemia of a similar etiology can also be caused by hemorrhagic diseases. The whole group is ill, when people have a tendency to become sick to bleeding through impaired homeostasis.

Symptoms and picture of blood in case of anemia as a result of hospitality of blood loss

key picture State of the art post-hemorrhagic anemia develops even more quickly. Before the main symptoms of sickness, you must admit to manifest a shock as a result of severe bleeding. In general, they are encouraged to:

• Reduction of a blood grip.
• Turbidity or loss of evidence.
• Strong bluishness, bluish appearance of the nasolabial fold.
• Thread-like pulse.
• Blues.
• Pitvischena pitlivist, and so the title of cold pit is promoted.
• Chills.
• Sudomi.

As soon as the bleeding goes into the distance, it is successful to zupiniti, then such symptoms change to the dullness, the noise in the vuhah, the second orintatsiya, the broken zoru, zadishkoy, the rhythm of the heart beat. Yak and earlier, it is possible to take advantage of the thinness of the curvatures and mucous membranes, lowering the arterial grip.

Here you will know the lecture about the methods of Lika

Anemia-Symptoms and Likuvannya https://youtu.be/f5HXbNbBf5w iron

A normal video can be seen in the report.

About chapter 19.08 itself.

About chapter 19.08 itself.

Doctor Komarovskiy to explain what causes an

Subscribe to the channel "About naygolovnishe" ▻ https: //www.y

Instagram: https://www.instagram.com/dr.philipp VK: https://vk.com/doctorphil

Subscribe to the channel "About naygolovnishe" ▻ https: //www.y

Subscribe to the channel "About naygolovnishe" ▻ https: //www.y

Anemia is a tse stan, which is to be promoted by practical

Hemolytic anemia - the price of anemia, which develops in the river

At the end of the video Torsunov Oleg Gennadiyovich talk about when

http://svetlyua.ru/Anemia, lіkuvannya folk remediesіhttp: // sve

Good day, dear friends! You are a doctor-Nutricia

I'm in: INSTAGRAM http://instagram.com/julia__rain TWITTER https://twitter.com/JuliaRain4 VKONTAKT

Anemia or nedokriv'ya - the price of reducing the concentration of g

Yak lіkuvati anemia? It helped me with zalizodeficitis

Fatal deficiency anemia. Symptoms, Signs and Methods

Anemia is one of the best common reasons vipadi

Changes in the results of blood analysis for a long period of time for the elimination of bleeding and development of anemia are tightly linked to the mechanisms of compensation, which are "included" in the body in the blood of the great promise. You can distribute at the following stages:

• The reflex phase, as it develops into the first, dobu after blood loss. To fix the overrun and centralization of blood circulation, to improve the peripheral support of the sudin. At the same time, there is a decrease in the number of erythrocytes with normal values ​​of the concentration of hemoglobin and hematocrit.
• The hydremic phase is opposed to the other by the fourth add. At the judge, you have been drinking pozaklitinna ridina, in the oven, glycogenolysis is activated, so that glucose is added to the increase. Progressively, symptoms of anemia appear in the blood picture: the concentration of hemoglobin decreases, the hematocrit decreases. However, the value of the quantitative indicator, as it was earlier, was in the norm. During the activation of thrombotic processes, the number of thrombocytes decreases, and after an hour of bleeding, leukopenia is more likely to develop.
• Cystkovomozkov phase is repaired from the fifth day after bleeding. Lack of constant organ and tissue activation in blood formation processes. In addition to the decreased hemoglobin, hematocrit, trombocytopenia and leukopenia at the third stage, the decrease in the backbone number of erythrocytes is more important. When a blood smear is reached, the presence of young forms of erythrocytes is indicated: reticulocytes, one of erythroblasts.

Some changes in the picture of blood are described in the situations of problems for the life of the family.

Symptoms and diagnosis of anemia in chronic bleeding

Chronic post-hemorrhagic anemia for its symptomatology is similar to hypersensitivity, as it regularly causes slight bleeding to a deficiency of a microelement. Stretching out a lot of bloodshed of blood, lay down on the step of this gravity. Її vis-à-vis the deposit in the concentration of hemoglobin. The norm for choloviks won is 135 - 160 g / l, and for women 120 - 140 g / l. In children, it is important to grow in fallowness from 200 to 200, and up to 150 from adults.

Stage of posthemorrhagic chronic anemia Concentration of hemoglobin

• 1 (light) step 90 - 110 g / l
• 2 steps ( average gravity) 70 - 90 g / l
• 3 steps (important) below 70 g / l

On the cob stage, the development of the sickness of the patient should be skipped on easier to mess up, Migotinnya "fly" in front of ochima, p_vischenu stomlyuvanost. It is the name of the wickedness and sleazy.

At the other stage, before the symptoms are overwhelmed, a decrease in appetite, inodi nudity, diarrhea abo, navpaki, constipation, zadishka is achieved. When hearing the heart tones, there are noises in the heart that are characteristic of chronic post-hemorrhagic anemia. Change and the camp of the skirting crooks: the skirting grows dry, peeling. The squadrons of the company have sore and fiery fires. Hairy and nigtiv will burn.

The importance of the steps of anemia is manifested in the same and in the fingers of the fingers and at the same time, there are specific relish addictions, for example, the ailments are to repair the credit, to change the smell. Even more often, the stage of chronic post-hemorrhagic anemia is supressed with advanced disease, stomatitis.

Diagnostics of post-hemorrhagic anemia is based on the results of clinical blood analysis. Most characteristic for all types of anemia is a decrease in the amount of hemoglobin and erythrocytes, there is a decrease in the colonic indicator. The yogic value is in between 0.5 - 0.6. In addition, with chronic post-hemorrhagic anemia, there are types of erythrocytes (microcytosis and shizocyti).

Likuvannia of the anemia of massive blood loss

Persh for everything, I need to bleed. As soon as it is called, then it is necessary to put on the jgut, to press and bring the victim to the lick. About the internal bleeding of the blood, cyanosis and the loss of quality, there is a strong dryness in the mouth. At home minds, I’ll help people in such a camp, it’s not a matter of fact, because the internal bleeding is carried out only in the minds of the stationary.

It is necessary to renew the blood circulation in the term of the term. For the whole, rheopolyglyukin, hemodez, polyglyukin are used. Gostru blood loss can also compensate for transfusions of blood from the urinary tract of the Rh factor and blood groups. Obsyag blood transfusion to become 400 - 500 ml. If you come in, you should be carried out as quickly as possible, so as if you had to spend a lot of time, you can lead to a lethal result.

If the drug is stopped, I will be shocked and all the necessary manipulations are carried out to a standard treatment, as it is possible for the introduced drugs in the form of a well-developed life for storing the deficiency of vitamins and microelements. Zazvychay priznachayutsya ferrum lek, ferlatum, maltofer.

Change the normal picture of blood to be updated in 6 - 8 days, ale drugs for the normalization of blood formation, prodovzhuyut until pivrok.

Likuvannya of chronic post-hemorrhagic anemia

The first and most important stage in the therapy of post-hemorrhagic chronic anemia of the disease in the presence of a bleeding dilemma and that of usunenia. Add 10 - 15 ml of blood to add more to the body of the blood flow, as it was necessary for the whole day since the day.

It is carried out in a complex way for a sick person, like in a ligamentous order, including consultations of a gastroenterologist, proctologist, hematologist, gynecologist for women, endocrinologist. As soon as it became ill, it became the reason for the development of chronic post-hemorrhagic anemia, it was inaccurately repaired.

At the same time, lykarsky drugs are recognized, as they take place. For the older yogo dobova dose get close to 100 - 150 mg. It is meant to be complex, as it can be used to replace ascorbic acid and vitamins in groups, which can be easily acquired. Tse sorbifer durules, ferroplex, fenuls.

In case of severe interruption of posthemorrhagic chronic anemia, transfusion of erythrocyte mass and injections is indicated for stimulation of the processes of blood formation. lykarsky zasobiv with zalizom. I mean Ferlatum, maltofer, likferr and other medicines.

Updating the main course of education

The triviality of the intake of drugs in the form of a drug. Besides the storage of new medicinal preparations for the renewal of the normal production of organs in the acidic and the renewal of the reserves in the body, it is even more important to improve the consumption.

Among the ratsioni people, who have suffered post-hemorrhagic anemia, in the general order, they are guilty of the presence of children and zalizo. The courier needs to be given meat, eggs, fermented milk products... Leaders behind the bulk of the meat є meat offal, especially yalovicha pechinka, meat, riba, іkra, beans, gorіkha, greek and groats.

When folded children respect, it is necessary to take the animals not just for those who want to take revenge on that product, but on the steps of their moisture in the body. It grows with the implantation of vegetables and fruits, such as the introduction of vitamins in and out of citrus fruits, black currants, raspberries and other fruits.

Excessive and post-hemorrhagic anemia therapy in children

Post-hemorrhagic anemia in children is more important, especially the form. The clever picture of the central pathology is practically not seen as an older person, but it is less developed more. And if in the older people the debilitated blood loss is compensated for by these reactions to the body, then in the child it can lead to a lethal result.

Likuvannya State Hotel chronic form post-hemorrhagic anemia in children of the same. If the cause of bleeding is revealed, it is indicated that the transfusion of erythrocyte mass with a dose of 10 - 15 ml per kg of vagi, preparations of zaliza is indicated. I will grow up individually in fallowness from the degree of severity of anemia and become a child.

For children at the time, it is close to the pivrok to recommend early introduction of complementary foods, and moreover, to repair the next few products with the addition of a large amount of food. For babies, the testimony was transferred to special vitamins. As soon as he got sick, he called for the development of post-hemorrhagic anemia, to be chronic and not to suffer, then the prophylactic course of the preparation in the hair must be repeated regularly.

With a quick ear of drinking and non-critical consumption of blood, the prognosis is generally friendly. To compensate for the deficiency of the child's wife shvidko on the mend.